effect of electromagnetic field on fetal cardiac output

The use of cellular [mobile] phone has been exploded world wide during last decade. Their rapid adoption by general public has resulted in an increased research interest in possible harmful health effects[Tahavanainen et al., 2004], This common use of cellular phones had, given rise to concerns about the potential influences of electromagnetic fields on human physiology [Croft et al., 2002]. We studied the effect of electromagnetic fields emitted by cellular phone on fetal heart rate patterns, end diastolic volume, end systolic volume stroke volume and cardiac output. Our results showed that after use of mobile phones on different gestational ages [group I, II and III], significant increase in FHR and significant reduction of EDV, ESV, SV and Cop. The percent of reduction in EDV was more than the percent of reduction in ESV in all groups. This prominent decrease in EDV more than the decrease in ESV suggests that the decrease in SV is mainly due to decrease in contractility. We conclude that the use of mobile phone has a negative effect on. Fetal heart so we recommend avoidance of cellular phone use especially in the early weeks of gestation and f necessary, the phone call should be less than ten minutes

role of angiotensin converting enzyme, prostaglandins and nitric oxide in acute renal failure and possibility of their interaction

Acute renal failure has remained a common and serious syndrome associated with high morbidity and mortality. The aim of the present work was to study the role of angiotensin converting enzyme, prostaglandins and nitric oxide in acute renal failure and to investigate the possibility that the renoprotective action of angiotensin converting enzyme inhibitors is mediated through prostaglandins or nitric oxide. The results of this work showed that angiotensin converting enzyme inhibitor [perindopril] reduced the glycerol induced renal failure in rats proving that angiotensin converting enzyme has a role in the pathogenesis of this condition. On the other hand, injection of the prostaglandin synthesis inhibitor [voltaren] caused non significant increase of the glyceral-induced acute renal failure this means that prostaglandins has no role in the protection against or pathogenesis of acute renal failure. This work showed, also, that injection of the nitric oxide synthesis inhibitor [L-NAME] aggravates the glycerol-induced acute renal failure this means that nitric oxide has a renoprotective effect. This study demonstrates, also, that injection of prostaglandin synthesis inhibitor [voltaren] or the nitric oxide synthesis inhibitor [L-NAME] together with the angiotensin converting enzyme inhibitor attenuates or even completely abolished its renoprotective effect, this gives a good evidence that prostaglandins and nitric oxide mediate the renoprotective action of the angiotensin converting enzyme inhibitors

study on the effect of prostaglandin F2 alpha on blood glucose in normal, diabetic and stressed rats

The aim of the present work was to study the effect of prostaglandin F2 alpha on blood glucose level in normal and diabetic rats and to study its protective effect against stress-induced hyperglycemia and alloxan-induced diabetes. The results obtained showed that injection of prostaglandin F2 alpha reduced the ether stress-induced hyperglycemia, while it has no significant effect on plasma glucose level in normal and diabetic rats. The results showed that it has no protective effect against alloxan-induced diabetes. Interpretation of these results suggested that prostaglandin F2 alpha might act through stimulation of insulin release