ABSTRACT
No abstract available.
Subject(s)
Female , Humans , Infant , Granulosa Cell Tumor , Granulosa CellsABSTRACT
Recently, we experienced two cases of tuberous sclerosis, in 4-year-4-month-old female and 17-month-old male patient. The diagnoses were established by typical clinical manifestations and brain CAT. Especially, in latter case, calcific density was successfully detected by brain CAT, while plain radiographs of the skull revealed still no evidence of calcification. A brief review of related literatures was made.
Subject(s)
Animals , Cats , Female , Humans , Infant , Male , Brain , Diagnosis , Skull , Tuberous SclerosisABSTRACT
We experienced a case of transient complete A-V block, which developed probably due to acute myocarditis of viral etiology. The patient was 13 year old boy, and was admitted to our ward with chief complaints of intermittent abdominal pain and chest discomfort. The auscultation of the heart revealed, the 1st heart sounds varied in intensity, and the E.K.G. findings, checked then, showed complete A-V block with atrial rate of 110/min and ventricular rate of 73/min. We recommended absolute bed rest and oral prednisolove(50mg/day). He discharged on 10th hospital day in good condition with normalized E.K.G. finding.
Subject(s)
Adolescent , Humans , Male , Abdominal Pain , Auscultation , Bed Rest , Heart , Heart Sounds , Myocarditis , ThoraxABSTRACT
We have experienced a case of 18p- syndrome in 22 month old girl who had craniofacial anomaly such as microcephaly, hypertelorism, nystagmus, depressed nasal bridge, complete bilateral cleft palate, both ear canal stenosis and mental retardation. Chromosome culture revealed short arm deletion of chromosome 18 in E group. As far as we know, this case is the first report of 18p- syndrome in Korea and brief review of related literature was made.
Subject(s)
Female , Humans , Infant , Arm , Chromosomes, Human, Pair 18 , Cleft Palate , Constriction, Pathologic , Ear Canal , Hypertelorism , Intellectual Disability , Korea , MicrocephalyABSTRACT
The bilirubin formed in utero by the fetus can cross the placena and be excreted by the maternal liver. After birth, however, the infant must excrete its own bilirubin and hepatic clearance is not sufficient to prevent its accumulation during first week of life This transition has been physiologic jaundice and defined as an elevation of serum bilirubin that does not exceed 12mg% that occurs in first week of life in the absence of hemolytic disease. It is becoming increasingly apparent that several factors act in concert and that no single mechanism is soly responsible for producing physiologic jaundice in the new born. A simple micromethod technique for the analysis of small samples has made it possible to record the changes in the level of bilirubin in the serum by means of daily observation a large number of infants. So, serial observations for serum bilirubin concentrations on 40 healty fullterm infants from birth to 7 day. The study revealed that 14 infants(38%) exhibited the bilirubin levels of 12mg% or higher. The means of the peak bilirubin concentrations were obtained and revealed most higher on the forth dey. Positive correlations were found between the intensity and the duration of physiologic icterus, between the degree of hyperbilirubinemia and the age at which the process attains its maximum intensity and between the maximum height of the bilirubin level and the total duration of elevated levels of bilirubin in the serum. The mean total bilirubin level of the physiologic jaundice in the Korean new born infants was remarkably higher than those observed in the white iufants. So, the definition of physiologic jaundice by zeuzer is not of use in the Korean full term in fants.
Subject(s)
Humans , Infant , Bilirubin , Fetus , Hyperbilirubinemia , Jaundice , Liver , ParturitionABSTRACT
Nine cases of paroxysmal supraventricular tachycardia were observed in the pediatric department of SNUH for the last 4 years from 1976. Brief presntatioen of the cases and review of the literatures were done.