ABSTRACT
Primary hyperparathyroidism is characterized by an increase in the secretion of parathyroid hormone (PTH), which causeshypercalcemia, determining, in varying degrees, the involvement of various organs and systems, increasing both maternaland fetal morbidity and mortality and compromising the prognosis of the binomial.We present the clinical case of a 28-year-old patient, primigravida, with a personal history of bilateral renal lithiasis,repeated pyelonephritis and a presumably nonfunctioning thyroid nodule. At 17 weeks ́ gestational age she is admitted forPRGHUDWH DQHPLD D PRQWK RI HYROXWLRQ SRRUO/ WROHUDWHG 3K/VLFDO H[DPLQDWLRQ ÀQGV QRGXOH LQ WKH WK/URLG ORGJH RI UDSLG JURZWK /DERUDWRU/ SURÀOH UDLVHV K/SHUFDOFHPLD DQG LQFUHDVHG LQWDFW 37+ PDNLQJ GLDJQRVLV RI SULPDU/ K/SHUSDUDWK/URLGLVP Surgical resection is performed at 24 weeks ́. The pathologic diagnosis was cystic parathyroid adenoma. In the evolutionshe presented hypothyroidism with normal levels of serum calcium and PTH. Being treated with calcium, calcitriol and T4.At 39 weeks labor begins spontaneously and a cesarean section is performed with diagnosis of non-reassuring fetal status,obtaining a vigorous newborn, with appropriate weight for gestational age. Good postpartum evolution