ABSTRACT
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ABSTRACT
Noncompaction ventricular myocardium is an unusual cause of cardiomyopathy. It is association with congenital heart defects, most often with outflow obstructive lesions or coronary anomalies. However, no factor could explain the arrest of development of myocardial structure (isolated form). The pathogenesis of isolated noncompaction is thought to be an arrest in endomyocardial morphogenesis. It has been reported that myocardial noncompaction could present as acquired disease. The most common site of involvement is the left ventricle, with right ventricular involvement being reported in a few cases. In this report, we present a case with noncompaction of the right ventricle (RV). Cardiac computed tomography angiography and magnetic resonance imaging demonstrated morphological abnormalities of the RV.
Subject(s)
Angiography , Cardiomyopathies , Heart Defects, Congenital , Heart Ventricles , Isolated Noncompaction of the Ventricular Myocardium , Magnetic Resonance Imaging , Morphogenesis , Multidetector Computed Tomography , MyocardiumABSTRACT
No abstract available.
Subject(s)
Female , Humans , Male , Cardiac-Gated Imaging Techniques , Coronary Vessel Anomalies/diagnostic imaging , Sinoatrial Node/diagnostic imaging , Tomography, X-Ray Computed/methodsABSTRACT
No abstract available.
Subject(s)
Female , Humans , Coronary Sinus/abnormalities , Tomography, X-Ray ComputedABSTRACT
No abstract available.
Subject(s)
Humans , Carpal Tunnel Syndrome/diagnostic imaging , Electromyography , Fingers/innervation , Median Nerve/diagnostic imaging , Ultrasonography, DopplerABSTRACT
We reviewed cases of primary colorectal adenocarcinoma to document synchronous colon and rectum adenocarcinoma SCRC. In a retrospective setting, 764 cases underwent surgical resection for primary colorectal adenocarcinoma and referred to the Department of Surgical Pathology, Uludag University, Medical Faculty for diagnoses between 1997 and 2006, were reviewed. Tumor site, depth of invasion, coexistence of adenoma, distance between these multiple primary tumors, degree of p53 expression, and p53 expression pattern indicating polyclonal or monoclonal origins were noted in order to establish a possible effect on prognosis. There were 28 cases with SCRC of colon and rectum. Nine cases were female, 19 cases were male female to male ratio was 1:2. Most of the cases were within the 5th and the 6th decades. There were statistically significant relationships between p53 expression and differentiation status p=0.001, and invasion depth p=0.03. Forty of 62 colorectal carcinomas showed immunohistochemical positivity for p53. Six cases showed a discordant pattern of p53 mutation among individual lesions indicating polyclonal origin. Synchronous colon and rectum adenocarcinoma is not rare. The incidence is 3.6% in our series. We believe that further studies with larger series are needed for p53 to prove useful in predicting prognosis of SCRCs and assessing the polyclonal origin of SCRC at a genetic level