ABSTRACT
No abstract available.
Subject(s)
Arteriovenous Fistula , Cerebral Infarction , Intracranial Embolism , Ultrasonography, Doppler, TranscranialABSTRACT
Progressive multifocal leukoencephalopathy (PML) is a demyelinating disease of the central nervous system caused by the human polyomavirus JC virus. PML mainly occurs in immunocompromised patients. A 36-year-old man with no evidence of immunosuppresion presented seizures. MRI scans of brain showed multifocal lesions in the cerebral white matters. JC virus DNA was positive in the cerebrospinal fluid examined by JC virus PCR. We report a rare case of PML presenting as viral encephalitis that occurred in a healthy adult person.
Subject(s)
Adult , Humans , Brain , Central Nervous System , Demyelinating Diseases , DNA , Encephalitis, Viral , Immunocompromised Host , JC Virus , Leukoencephalopathy, Progressive Multifocal , Magnetic Resonance Imaging , Polymerase Chain Reaction , Seizures , VirusesABSTRACT
No abstract available.
Subject(s)
Cerebral Infarction , Fingers , Infarction , Muscle WeaknessABSTRACT
BACKGROUND: Common carotid artery intima-media thickness (IMT) is an accepted sonographic marker of early atherosclerosis, and increased IMT represents a risk of ischemic stroke. The purpose of this study was to evaluate the correlation between common carotid artery IMT and stroke risk factors in patients with ischemic stroke. METHODS: Subjects were 381 patients with acute ischemic stroke. High resolution B-mode ultrasonography was performed to measure IMT at common carotid artery. Multivariate regression analysis was performed to determine the independent factors related to common carotid artery IMT. RESULTS: The variables that strongly predicted an increase in the common carotid artery IMT were age (P <0.001) and diabetes mellitus (P <0.001). CONCLUSION: Increased common carotid artery IMT is independently associated with age and diabetes mellitus in patients with ischemic stroke.
Subject(s)
Humans , Atherosclerosis , Carotid Arteries , Carotid Artery, Common , Diabetes Mellitus , Risk Factors , StrokeABSTRACT
Hypoxic brain damage after carbon monoxide (CO) occurs predominantly in the basal ganglia, cerebral white matter and cerebral cortex. CO poisoning is complicated by various neurological manifestations as well as hemorrhagic infarction. Acute cerebral thromboembolic infarction following CO poisoning is very rare. We experienced a case of thromboembolic infarction in the middle cerebral artery territory occurred in a healthy young adult without any risk factor for stroke during treatment for hypoxic encephalopathy caused by acute CO poisoning.
Subject(s)
Humans , Young Adult , Basal Ganglia , Carbon , Carbon Monoxide , Carbon Monoxide Poisoning , Cerebral Cortex , Cerebral Infarction , Hypoxia, Brain , Infarction , Middle Cerebral Artery , Neurologic Manifestations , Risk Factors , StrokeABSTRACT
Calcium deposits in the basal ganglia and in the other areas of the brain may occur as idiopathic disease or secondary to metabolic disturbances including hypoparathyroidism. Fahr's syndrome is characterized by secondary cerebral calcification and variable clinical manifestations of movement disorder, cognitive impairment, and behavioral changes. We report a 48 year-old man with Fahr's syndrome due to idiopathic hypoparathyroidism who presented with recurrent epileptic seizures. Brain CT showed multiple calcifications of the Fahr type in the basal ganglia, thalami, cerebellar white matter and dentate nuclei, and centrum seiovale. Brain MRI diffusion-weighted image showed high signal intensity in the right anterior temporal region.