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1.
Asian Journal of Sports Medicine. 2011; 2 (3): 134-144
in English | IMEMR | ID: emr-163014

ABSTRACT

The aim of this study was to assess the effects of Ramadan fasting on several biochemical and anthropometric parameters in physically active men by comparing fasters and nonfasters before, during, and after Ramadan. Eighteen physically active men [10 fasters and 8 nonfasters] participated in this study. Subjects visited the laboratory for a total of four sessions on the following occasions: three days before Ramadan [Bef-R], the 15th day of Ramadan [Mid-R], the 29th day of Ramadan [End-R], and 21 days after Ramadan [Post-R]. During each session, subjects underwent anthropometric measurements, completed a dietary questionnaire and provided a fasting blood sample. Body weight and body fat percentage decreased in fasters by 1.9% [P<0.001] and 6.2% [P=0.003], respectively, but increased in nonfasters by 2.2% [P<0.001] and 10.2% [P=0.001], respectively, from Bef-R to End-R. Fasters' hematocrit and hemoglobin increased by 5.3% [P<0.001] and 6.3% [P=0.01], respectively, from Bef-R to End-R, while neither of these parameters changed in nonfasters. Fasters experienced an increase in the following parameters from Bef-R to End-R: urea [8.7%; P<0.001], creatinine [7.5%; P<0.001], uric acid [12.7%; P<0.001], serum sodium [1.9%; P<0.001], serum chloride [2.6%; P<0.001] and high-density lipoprotein cholesterol [27.3%; P<0.001]. Of these parameters, only creatinine increased [4.4%; P=0.01] in nonfasters. We conclude that Ramadan fasting lowers body weight and body fat percentage and can elevate high-density lipoprotein cholesterol in physically active men. However, practicing aerobic exercise during a hot and humid Ramadan month can induce a state of dehydration marked by an increase in some renal function markers and serum electrolytes

2.
Tunisie Medicale [La]. 2007; 85 (7): 610-613
in French | IMEMR | ID: emr-139311

ABSTRACT

The aim of this work was to study the clinical and therapeutic features of this affection. Our retrospective study concerned two cases of hyperlipemic pancreatitis treated during 6 years from 1998 to 2003. The incidence of this affection was 1.42%. Our patients were respectively a man aged 32 years without any significant history and a pregnant woman aged 24 years with a positive history of dyslipidemia. Clinical, biologic and radiological data didn't differ from those of other causes of acute pancreatitis. The hyperlipemic origin was confirmed by a lactescent serum and a rate of triglycerides greater than 10 g/1. The course was characterized by the recurrence of pancreatitis related to the increased triglycerid serum level over 10 g/1. Plasmapheresis and administration of heparin and/or insulin seem to be efficient in reducing the serum level of triglyceride and in improving the course. Hygieno-dietary measures and hypolipemiant treatment were necessary for the level of triglyceirdes in serum below 10 g/1. We in the absence of comolicatons, surgery seemed umnecessry in the two cases. the clinical pattern of acute hyperlipemic pancreatitis doesn't include any specific elements, but its treatment and prevention must take into account the associated hyperlipidemia

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