arthrites reactionnelles

Reactive arthritis are definied as steriles arthropathies using classic bacteriological techniques. They are due to extra articular infection and are often associated with HLA B27. the outcome of these arthritis is caracterised by the recurrence of flares with sometimes appearition of ankylosing spondylitis. The pathogenesis of reactive arthritis is modified when bacterial antigens or alive micro-organisms are discovered in involved joints. Several current works have underlined the interest of antibiotic therapy in the chlamydial reactive arthritis. Chronic forms can justify the use of anti-rheumatic drugs such as salazopyrine

Manifestations musculo-squelletiques de l'hypothyroidie

Rheumatologic manifestations are varied during the hypothy-roidism. They can be articular, abarticular, muscular or osseous, consequences oF an inappropriale hormonal secretion. Their diagnosis is easy when hypolhyroidism is known, but they can be inaugural. Their evolution is favorable under substitute treatment

Manifestations rhumatologiques de l'infection a parvovirus B19

Rheumatologic manifestations of parvovirus B19 infection is a recent individualization. They are rather frequent and varied and meet at the young adult's. generally, it is responsible for acute, bilateral and symmetrical arthritis, usually involving distal joints and somrtimes associated to signs caused by viral infections. Prolonged articular forms were described but are rare being able to sometimes feign a nocive rheumatoid arthritis because of their clinical aspect and of a seropositivity. Axial manifestations were also repoted. The diagnosis of the recent infection by the parvovirus B19 can be confirmed by ELISA identification of specific IgM antibodies. The mechanism of the articular manifestations is still unknown and the link between parvovirus B19 and systemic vasculitis is questioned

Association lupus erythemateux systemique - hypothyroidie. A propos d'un cas

The association of systemic lupus erythematosus [SLE] and hypothyroidism is uncommon, and concerns essentially the autoimmun thyroiditis. However, the coexistence SLE-primary hypothyroidism is exceptional. We report an observation of 25-old woman, who presents a clinical form of hypothyroidism, confirmed by hormonal tests, and which is associated lo polyarthritis and proximal myopathy non ameliorated by opo-therapy. The explorations conclude in SLE. A favourable evolution of muscular and articular signs is obtained with cortico-therapy

Aspect pseudo-polymyositoque d'une sclerose laterale amyotrophique

We present three cases of short-coupled variant of torsade de pointes with review of the literature. These women presented with syncope or presyncope due to torsade de pointes initiated by a short-coupled premature ventricular beat and without evidence of prolonged QT. There were no electrolyte disturbances in all cases, no apparent structural heart disease in two cases and a mild interventricular septum hypertrophy in the other case. One patient took spiramycin and metro nidazole and another was taking pheniramin and lincomycin without any evidence of cause to effect relationship. One patient responded to verapamil but died suddendly after 44 months of follow-up. The two others. Received implantable cardioverter-defibrillators and verapamil per os. They still alive 46 and 54 months later. Short-coupled variant of torsade de pointes have a high incidence of sudden death, so it is very important for physicians to identify and treat it promptly, long-term verapamil treatement is effective but still insufficient and patients should be considered for implantable cardioverter-defibrillator therapy