ABSTRACT
Coronary heart disease is an increasing illness and one of the gold standards for their diagnosis is catheterization [CA]. Post-CA bed rest may cause back pain. In Iran, there has been no study about the effects of changing the position on their back pain after coronary angiography. This study was designed to explore these effects amongst the patients admitted to Taleghani hospital, 2006-2007. This clinical trial was carried out on 130 patients hospitalized for coronary angiography. Each Patient was assigned randomly to either the control group [which remained in supine position for 6 hours after coronary angiography], or experimental group. The position of experimental group were changed hourly, from supine to 30 degree elevation and semi position [elevated to 45 degrees] during the first 6 hours after coronary angiography. Check list and numeric pain intensity scale were used for data collection. None of patients developed bleeding, haematoma and arterial thrombosis. There were no significant difference between two groups. There was a significantly lesser pain intensity in experimental group [P<0.001]. The extention of back pain was lesser in experimental group but it was not significantly different [P<0.07]. Changing the bed position of patients would reduce the back pain without increasing the risk of vascular complications. It also promotes physical comfort among patients undergone coronary angiography
Subject(s)
Humans , Low Back Pain , Supine Position , PostureABSTRACT
In view of the high morbidity and mortality of patients with congestive heart failure, early diagnosis is of paramount importance. To assess the diagnostic and prognostic utility of plasma levels of the NT-Pro BNP in patients presenting with heart failure this study was carried out. This cross-sectional study was performed on patients with heart failure admitted in the cardiology ward of Taleghani Medical Center during a certain period from 2005- 2006. Subjects with acute coronary syndrome, hyperthyroidism, renal failure [GFR<60], severe valvular stenosis, severe pulmonary disease, ARDS, hepatic cirrhosis, primary hyperaldosteronism and the candidates for heart transplant were excluded from the study. Blood samples were collected and plasma levels of NT-Pro BNP were measured by ELISA method [Biomedica- crop, Bratislava Slovakia]. Results were documented and analyzed using SPSS software. Mann- Whitheny and KrusKall-Wallis tests were utilized for analysis. Values were analyzed for confounding variables [e.g age, sex, blood pressure, and ischemic heart disease and NYHA class]. 79 patients full filled the criteria of inclusion. 70% were males and mean age of patients was 63 +/- 14 years. Mean plasma NT-Pro BNP level was 421 +/- 387. In subjects <60 years of age, the mean levels were 309 +/- 300, and among those >/= 60 years it was 486 +/- 419, [P<0.05]. With advancing clinical stage, the mean plasma level of NT-Pro BNP increased 6 folds, [p<0.01], whereas with advancing NYHA class it increased 4 folds [P<0.01]. It seems that plasma NT-Pro BNP level is a suitable indicator of the severity of heart failure