ABSTRACT
This case report, along with the review presented, describes a patient diagnosed with acute viral hepatitis, who developed a framework of intense abdominal pain and laboratorial alterations compatible with acute pancreatitis. The association of acute pancreatitis complicating fulminant and non-fulminant acute hepatitis virus (AHV) has been reported and several mechanisms have been proposed for this complication, but so far none is clearly involved. As acute hepatitis is a common disease, it is important to stimulate the development of other studies in order to determine local incidence and profile of patients presenting this association in our environment.
Este relato de caso, junto com a revisão de literatura, descreve um paciente com diagnóstico de hepatite viral aguda, que desenvolveu quadro de dor abdominal intensa e alterações laboratoriais compatíveis com pancreatite aguda. Casos de pancreatite aguda complicando hepatites virais agudas fulminantes e não fulminantes tem sido esporadicamente relatados e vários mecanismos são propostos para explicar esta complicação, no entanto sua causa ainda se mantém desconhecida. Como a hepatite aguda é doença comum, é importante estimular o desenvolvimento de mais estudos na América Latina que visem determinar a incidência local e o perfil dos pacientes que apresentam esta complicação.
Subject(s)
Adult , Female , Humans , Hepatitis, Viral, Human/complications , Pancreatitis/etiology , Acute Disease , Hepatitis, Viral, Human/classification , Hepatitis, Viral, Human/diagnosis , Pancreatitis/diagnosisABSTRACT
Based on the idea that the pathogenic-etiologic mechanism is the " blockadeof liver channels, blood stasis and Qi stagnation resulting in malnutrition of liver due to iack of blood supply", the treating principle of " e-liminating stasis and supporting body resistance" was established with the combined use of extracts from peach -kernel plus cordicep hypha. 6 cases were thus treated. All of them received laparoscopy , and liver biopsy before and after the treatment. The gross appearance, histology and ultra. - microscopic structure and irnmuno - histo - chemistry were examined. Results showed that most of the activatedfat - storage cells after treatment were silent the I, III types of collagen depo - sited among the spaces of peri - sinusoid and liver cells and FN, LM weredecreased in part of the patient, resulting in less intrahepatic fibrosis, degradation of collagen fibres deposited in inter hepatic cellular spaces, inhibition and reversion of capillariza-tion oi liver sinusoid, hence, improvement of degener-ation of liver cells in most cases. The texture of the liver were turning normal with amelioration of portal hypertension and decrease of varicose mesenteric ves-seis. The liver turned red, swelling of falciform lig(?)-ment disappeared, and ascitis subsided.