ABSTRACT
No abstract available.
Subject(s)
Middle Aged , Male , Humans , Liver/pathology , Hepatitis, Alcoholic/pathologyABSTRACT
No abstract available.
Subject(s)
Aged , Humans , Male , Amiodarone/adverse effects , Anti-Arrhythmia Agents/adverse effects , Hepatitis, Alcoholic/diagnosis , Chemical and Drug Induced Liver Injury, Chronic/diagnosisABSTRACT
No abstract available.
Subject(s)
Humans , Chronic Disease , Hepatocytes/ultrastructure , Inclusion Bodies/ultrastructure , Liver Diseases/pathologyABSTRACT
A case of micronodular cirrhosis of the alcoholic type developed following an intestianl bypass surgery in a 47 year-old nonalcoholic male patient is presented. The patient denied any drug use of a long duration and had no diabetes mellitus. Five years before, a side to side ileo-transverse colon anastomosis had been performed for perforated intestinal tuberculosis at 1 m proximal to the ileocecal valve, bypassing a short segment of ileum (about 1.5 m) and transverse colon. The ileum distal to the perforated site had been found completely stenosed. He was severely lean with evidences of nutritional deficiency such as low serum levels of the albumin and vitamin B12. The liver biopsy showed a fatty change, Mallory bodies and perihepatocellular collagenosis within the cirrhotic nodules. The present case suggests that, when there are blind loop formation and nutritional deficiency, hepatic changes identical to those following jejunoileal bypass could develop even by reduction of a shore segment of the small intestine.
Subject(s)
Male , Humans , BiopsyABSTRACT
This is to describe a neonatal hepatitis with pericellular hepatic fibrosis and Mallory bodies in a sero-positive infant for IgM anti-CMV. A necropsy of the liver revealed severe heaptocellular swelling with many intracytoplasmic hyaline bodies, pronounced fibrosis of a creeping type, bile stasis with ductular proliferation, and the lack of parenchymal regeneration. These microscopical changes of the liver resembled those of Indian Childhood Cirrhosis (ICC). In the present case the patient's serum IgM anti-CMV is the only clue for the etiological diagnosis.