ABSTRACT
Se revisan la fisiopatología y los mecanismos que producen la sofocación en el hematoma cervical y se determina que el único tratamiento con posibilidades de éxito vital es el inmediato drenaje del hematoma.
In this article are reviewed the physiopathology and the mechanisms that causes suffocation in the cervical hematoma. It is determined that the only treatment with possibilities of vital success is the immediate drainage of the hematoma.
Subject(s)
Humans , Asphyxia/etiology , Drainage , Hematoma/physiopathology , Hematoma/therapy , Postoperative Complications , Asphyxia/physiopathology , Cervical Vertebrae/surgery , Neck/surgeryABSTRACT
PURPOSE: The usefulness of body movements that occur during sleep when assessing perinatal asphyxia and predicting its long-term consequences is contradictory. This study investigated whether neonatal rats manifest these movements in compensatory rebound after asphyxia, and if these alterations play an important role in its pathogenesis. METHODS: Eight neonatal rats (aged 6-48h) were implanted with small EMG and EKG electrodes and sleep movements were recorded over a 30-minute control period. Recordings were continued during asphyxia caused by the enclosure of the animal in a polyvinyl sheet for 60 minutes, followed by a 30-minute recovery period. RESULTS: Heart rate was lowered to bradycardic level during asphyxia causing behavioral agitation and increased waking time during the initial phase (30 minutes). Sleep-related movements were also significantly reduced from 12.5 ± 0.5 (median ± SE/2min) to 9.0 ± 0.44 in the final half of the period (Anova, p<0.05). Movement frequency increased in the recovery period to 15.0 ± 0.49 (Anova, p<0.05). CONCLUSION: These data show that newborn rats present compensatory rebound of body movements during sleep which may help in the diagnosis of asphyxia and other problems related to sleep parameters.
OBJETIVO: A utilidade dos movimentos corporais (MC) que ocorrem durante o sono para diagnosticar e predizer as conseqüências, em longo prazo, da asfixia perinatal é contraditório. Este estudo investigou se ratos recém-nascidos (RN) manifestam MC em resposta compensatória à asfixia, e se estas alterações podem ter alguma importância na sua patogênese. MÉTODOS: Oito ratos RN (6-48h de vida) foram submetidos à implantação de pequenos eletrodos para registros da eletromiografia e eletrocardiografia. Os MC e a freqüência cardíaca (FC) foram registrados durante períodos de 30 min: fase controle (F1), fases de asfixia (F2; F3) e fase de recuperação pós-asfixia (F4). A asfixia foi promovida pelo envolvimento completo do animal com uma lâmina de polivinil. RESULTADOS: A FC diminuiu progressivamente durante F2 e F3 até a bradicardia. Em F2 houve grande agitação dos animais e aumento dos períodos de vigília. Em F3 houve redução significante dos MC de 12,5 ± 0,5 (Md ± SE/2min) para 9,0 ± 0,44 (P<0,05). A freqüência dos MC aumentou em F4 para 15,0 ± 0,49. CONCLUSÃO: Estes dados mostram que ratos RN com asfixia apresentam MC compensatórios durante o sono que podem ajudar no diagnóstico desta afecção e de outros problemas relacionados aos parâmetros do sono.
Subject(s)
Animals , Rats , Asphyxia/physiopathology , Motor Activity/physiology , Parasomnias/physiopathology , Sleep/physiology , Animals, Newborn , Asphyxia/complications , Disease Models, Animal , Electromyography , Heart Rate , Polysomnography , Parasomnias/etiology , Rats, Wistar , Time FactorsABSTRACT
OBJECTIVE@#To explore the role of diaphgram fatigue in the death from hanging with bound upper limbs of rabbits.@*METHODS@#Rabbits were hanged with upper limbs bound, then the data of EMGdi were gathered@*RESULTS@#By analyzing power spectral of EMGdi in experiment, we compare the ratio change of H/L between pre-experiment and post-experiment. There is a significance decrease of the ratio of H/L, so it indicates that diaphgram fatigue does exist.@*CONCLUSION@#Diaphgram fatigue plays an important role in the death from hanging with limbs of rabbits bound.
Subject(s)
Animals , Female , Male , Rabbits , Asphyxia/physiopathology , Cause of Death , Diaphragm/physiopathology , Electromyography , Forensic Medicine , Muscle FatigueSubject(s)
Humans , Female , Pregnancy , Infant, Newborn , Anesthesia, Conduction , Anesthesia, Conduction/statistics & numerical data , Anesthesia, Conduction , Asphyxia/physiopathology , Fetal Distress/diagnosis , Fetal Distress/etiology , Fetal Distress/mortality , Fetal Distress/physiopathology , Fetal Distress/therapy , Cesarean Section/classification , Cesarean Section , Emergency Treatment , Hypoxia/prevention & controlSubject(s)
Humans , Child , Asphyxia/diagnosis , Asphyxia/drug therapy , Asphyxia/etiology , Asphyxia/physiopathology , Asphyxia/rehabilitationABSTRACT
Introducción. la asfixia traumática se caracteriza por cianosis cervical y facial, hemorragia subconjuntival, ingurgitación vascular grave y equimosis de cara y cuello. Estos signos se cree son complicaciones asociadas a este síndrome son neurológicas, consistentes en agitación y desorientación inicial; y pulmonares, que pueden ser desde edema pulmonar leve hasta hemorragia parenquimatosa extensa. El pronóstico es bueno y la mortalidad y morbilidad pueden ser resultante de lesiones asociadas. Casos Clínicos. Se presentan 5 niños con asfixia traumática. Todos los pacientes fueron consecutivos a trauma compresivo en el tórax. Uno de ellos tenía síndrome de SCIWORA. La contusión pulmonar fue una lesión comúnmente asociada. Todos sobrevivieron. Conclusiones. La asfixia traumática es un evento raro y su diagnóstico es relativamente fácil. Frecuentemente, requiere sólo de medidas sintomáticas y el pronóstico es bueno
Subject(s)
Humans , Male , Female , Asphyxia/complications , Asphyxia/physiopathology , Pediatrics , Thoracic Injuries/complicationsABSTRACT
This study analyzes variations of oxygen tension induced by asphyxia in the myocardium (MpO2) and coronary sinus blood (CSpO2) of the anesthetized open-chest dog. Oxygen tension was measured polarographically and arterial blood pressure and coronary blood flow were recorded simultaneously. Asphyxia increased arterial pressure and coronary blood flow but decreased both MpO2 and CSpO2. During the reoxygenation phase, arterial pressure and coronary flow decreased gradually. Reoxygenation induced a rapid but transient increase in CSpO2 above control levels. In contrast, MpO2 returned gradually to the basal state. The mechanisms(s) involved in the differential effects of asphyxia followed by reoxygenation on MpO2 and CSpO2 remain to be elucidated