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Article in Chinese | WPRIM | ID: wpr-879796


A boy, aged 2 years and 4 months, had a sudden onset of blepharoptosis of the right eyelid, accompanied by the mouth deviated to the right side, drinking cough, nystagmus, and developmental regression. Cranial MRI showed softening lesions formed after infarction of the right dorsolateral medulla oblongata, while head CT angiography showed no imaging of the proximal part of the V4 segment of the right vertebral artery. The child was diagnosed with dorsolateral medulla oblongata syndrome and was treated with gamma globulin to regulate immune function, with mannitol to reduce neuronal edema, with low-molecular-weight heparin sodium to improve local hypercoagulation of occluded blood vessels, with hyperbaric oxygen to improve local ischemia and hypoxia and promote the recovery of brain function, and with neuromuscular electrical stimulation to promote the recovery of neuromuscular function. Before discharge, only mild right ataxia and Horner syndrome remained. This article reports the first case of infantile dorsolateral medulla oblongata syndrome and provides experience for the diagnosis and treatment of the disease.

Blepharoptosis/etiology , Child, Preschool , Dysarthria/etiology , Humans , Lateral Medullary Syndrome/diagnosis , Magnetic Resonance Imaging , Male , Medulla Oblongata/diagnostic imaging
Rev. bras. ortop ; 54(5): 605-608, Sept.-Oct. 2019. graf
Article in English | LILACS | ID: biblio-1057931


Resumo The posterior inferior cerebellar artery usually arises from the intracranial segment (V4) of the vertebral artery. Despite its mean diameter of 2 mm, it usually irrigates important areas of the brain. When occluded, whether due to trauma or surgery, it may cause infarction in the brain stem and cerebellum. The present report describes a case of incidental finding of a posterior inferior cerebellar artery arising from the cervical segment (V3) of the vertebral artery, demonstrated by angiography. The findings were recorded and compared to those of earlier publications. Brief explanations regarding anatomy, vascular anomalies and embryology were provided. A literature review showed that anomalous branches of the cervical segment of the vertebral artery are infrequent andmust be known. A better understanding of anatomy and its variations enables an accurate topographic diagnosis, as well as the planning of the optimal surgical approach and therapy. Knowledge of this anatomical variation is essential because, if it is mistaken for a muscle branch and coagulated, this can cause ischemia and disabling sequelae.

Resumo A artéria cerebelar inferoposterior geralmente nasce do segmento intracraniano (V4) da artéria vertebral. Apesar de ter diâmetro médio de 2 mm, usualmente irriga áreas eloquentes do encéfalo. Quando ocluída, seja por trauma ou cirurgia, pode causar infarto no tronco encefálico e no cerebelo. Apresentamos um caso de artéria cerebelar inferoposterior com origem anômala no segmento cervical (V3) da artéria vertebral, demonstrado por angiografia. Os resultados foram registrados e comparados com os de publicações anteriores. Foram fornecidas breves explicações sobre a anatomia, anomalias vasculares e embriologia. A revisão da literatura mostrou que os ramos anômalos do segmento cervical da artéria vertebral são infrequentes e devem ser conhecidos. Uma melhor compreensão da anatomia e suas variações permite fazer um diagnóstico topográfico preciso, bem como planejar a abordagem e a terapia cirúrgicas ideais. O conhecimento dessa variação anatômica é essencial, pois, se confundida com um ramo muscular e coagulada, pode causar isquemia com sequelas incapacitantes.

Humans , Male , Middle Aged , Vertebral Artery , Lateral Medullary Syndrome , Brain Stem , Cerebral Angiography , Angiography , Anatomic Variation , Intraoperative Complications
Article in Korean | WPRIM | ID: wpr-766767


Opalski syndrome is a rare lateral medullary infarction variant presenting with ipsilateral motor deficits known to be caused by involvement of the post-decussating pyramidal tract. Here, we report two rare cases of Opalski syndrome presenting as ipsilateral sensorimotor deficits in cerebral infarction.

Cerebral Infarction , Infarction , Lateral Medullary Syndrome , Pyramidal Tracts
Rev. argent. neurocir ; 32(4): 222-229, dic. 2018. ilus, graf
Article in Spanish | LILACS, BINACIS | ID: biblio-1222531


Introducción: El recorrido del "loop subarcuato" de la arteria cerebelosa anteroinferior (ACAI) presenta múltiples variaciones que condicionan además su principal eferencia, la arteria subarcuata (ASA). El espectro de variaciones de este complejo ha sido referido en la literatura de forma inconexa y desorganizada. Material y Métodos: Se propuso una clasificación sistematizada de las variantes del complejo ACAI-ASA, basada en la interacción del hueso petroso y la ACAI en el periodo embrionario. La misma fue aplicada en una serie de pacientes estudiados mediante secuencia CISS (constructive interference in steady state) de resonancia magnética para categorizar las relaciones presentes en el ángulo pontocerebeloso (APC). Resultados: Se evaluaron 84 pacientes, incluyendo 161 APC. Todos los grados propuestos fueron identificados en la serie evaluada. Las proporciones encontradas en la gradación propuesta se mantuvieron en el rango de las publicaciones aisladas. Conclusión: La clasificación propuesta para el complejo ACAI-ASA permitió distinguir y objetivar consistentemente el espectro de variaciones.

Introduction: The pathway of the anterior inferior cerebellar artery's (AICA) "subarcuate loop" can vary extensively. This variability also affects its main branch, the subarcuate artery (SAA). The spectrum of variations observed with this combination of vessels is inadequately described in the literature. Methods and Materials: A systematized classification system for AICA-SAA complex variants was proposed, based upon interactions between the petrosal bone and the AICA in embryos. This classification scheme then was applied to a series of patients assessed by magnetic resonance CISS (constructive interference in steady state) sequences, to categorize the cerebellopontine angle (CPA) relationships. Results: Eighty-four patients were evaluated, encompassing 161 CPA. All the proposed grades were identified in the evaluated series. The proportions found with the proposed gradation system were within the range of previous publications. Conclusions: The AICA-SAA complex classification system that we proposed allowed for consistently distinguishing and objectifying the spectrum of variations seen in the subarcuate loop.

Humans , Lateral Medullary Syndrome , Arteries , Cerebellopontine Angle
Medicina (B.Aires) ; 78(5): 364-367, oct. 2018. ilus
Article in Spanish | LILACS | ID: biblio-976126


Presentamos dos casos de accidente cerebrovascular en sujetos con trayecto extracraneal de la arteria cerebeloso póstero-inferior. Caso 1: varón de 21 años, quien presentó ataxia y dismetría derecha luego de un traumatismo cervical en un partido de rugby. Caso 2: mujer de 56 años, quien inició con vértigo y hemiparesia izquierda luego de esfuerzo físico intenso. En ambos casos, los estudios angiográficos mostraron un trayecto extracraneal de la arteria cerebelosa póstero-inferior. Este vaso raramente se origina por debajo del foramen magno, en relación cercana con las primeras tres vértebras cervicales y la articulación atlanto-axial. En este nivel, está expuesta a daño mecánico causante de disección, como por ejemplo trauma directo, manipulación cervical abrupta o extensión cefálica prolongada. Por lo tanto, en pacientes con accidente cerebrovascular de región lateral de bulbo y trayecto extracraneal de la arteria cerebelosa póstero-inferior se debería considerar esta asociación.

We present two cases of lateral medullary stroke in subjects with extracranial trajectory of the postero-inferior cerebellar artery. Case 1: a 21-year-old male who presented ataxia and right dysmetria after cervical trauma in a rugby match. Case 2: 56-year-old woman, who started with vertigo and left hemiparesis after intense physical effort. In both cases, the angiographic studies showed an extracranial trajectory of the posterior inferior cerebellar artery. This vessel rarely originates below the foramen magnum, in close relationship with the first three cervical vertebrae and the atlanto-axial joint. At this level, it is exposed to mechanical damage causing dissection, such as direct trauma, abrupt cervical manipulation or prolonged cephalic extension. Therefore, this association should be considered in patients with stroke of the lateral region of the bulb and extracranial trajectory of the posterior-inferior cerebellar artery.

Humans , Male , Female , Middle Aged , Lateral Medullary Syndrome/etiology , Cerebellum/blood supply , Posterior Cerebral Artery/injuries , Aneurysm, Dissecting/complications , Lateral Medullary Syndrome/pathology , Lateral Medullary Syndrome/diagnostic imaging , Magnetic Resonance Imaging/methods , Cerebral Angiography/methods , Cerebellum/injuries , Cerebellum/pathology , Cerebellum/diagnostic imaging , Posterior Cerebral Artery/pathology , Posterior Cerebral Artery/diagnostic imaging , Aneurysm, Dissecting/pathology , Aneurysm, Dissecting/diagnostic imaging
Article in English | WPRIM | ID: wpr-714056


Hiccups are an involuntary contraction of the diaphragm that may repeat several times per minute. In general, hiccups are very common, transient, and self-limited. However, if the condition persists longer than days or months, it impacts a patient's quality of life. Pharmacologic and non-pharmacologic methods are used for the treatment of persistent or intractable hiccups. Nerve block and stimulation have been shown to be effective through neural pathway interruption or stimulation of the hiccup reflex arc. Stellate ganglion block (SGB) is an injection of local anesthetic adjacent to a group of nerves in the neck known as the stellate ganglion. The authors report a case of SGB as an effective treatment for a patient with intractable hiccups resulting from right lateral medullary syndrome.

Diaphragm , Hiccup , Humans , Lateral Medullary Syndrome , Neck , Nerve Block , Neural Pathways , Quality of Life , Reflex , Stellate Ganglion , Sympathetic Nervous System
Article in English | WPRIM | ID: wpr-717706


We describe the case of a patient who had infarction of the posterior inferior cerebellar artery (PICA) after a chiropractic cervical manipulation. A 39-year-old man visited the emergency room with signs of cerebellar dysfunction, presenting with a 6-hour history of vertigo and imbalance. Two weeks ago, he was treated by a chiropractor for intermittent neck pain. At the time of admission, brain computed tomography, magnetic resonance imaging, and angiography revealed an acute infarction in the left PICA territory and occlusion of the extracranial vertebral artery (VA; V1/2 junction) as a result of the dissection of the VA. Angiography revealed complete occlusion of the left PICA and arterial dissection was shown in the extracranial portion of the VA. He was treated with antiplatelet therapy. Three weeks later, he was discharged without any sequelae. The possibility of VA dissection should be considered at least once in patients presenting with cerebellar dysfunctions with a recent history of chiropractic cervical manipulation.

Adult , Angiography , Arteries , Brain , Cerebellar Diseases , Cerebral Infarction , Chiropractic , Emergency Service, Hospital , Humans , Infarction , Lateral Medullary Syndrome , Magnetic Resonance Imaging , Manipulation, Chiropractic , Manipulation, Spinal , Neck Pain , Pica , Vertebral Artery , Vertigo
Repert. med. cir ; 27(3): 177-179, 2018. Il., fotos
Article in English, Spanish | LILACS, COLNAL | ID: biblio-982031


El síndrome de Opalski es la presencia de hemiplejia ipsilateral asociada con síntomas del síndrome medular lateral. Diferentes estudios confirman que el infarto en tales casos ocurre a un nivel más bajo que el encontrado en el síndrome de Wallenberg. Se localiza debido a la presentación característica, el territorio único de suministro de sangre y el área pequeña de lesión. La implicación del tracto corticoespinal ipsilateral después de la decusación piramidal, o compresión de la decusación, podrían causarlo. Presentamos el caso de una mujer de 71 años que desarrolló hemiplejia izquierda y dificultad repentina para caminar, acompañada de diplopía, cefalea, disfagia y vómito recurrente. La imagen de resonancia magnética demostró un infarto lateral medular izquierdo. La angiografía reveló estenosis distal y proximal de la arteria vertebral izquierda e hipoplasia de la arteria vertebral derecha.

Opalski syndrome is the presence of ipsilateral hemiplegia which is associated with symptoms of a lateral medullary syndrome. Studies confirm that the infarction, in such cases, occurs at a lower level than that found in Wallenberg syndrome. This syndrome is localized because of characteristic presentation, the unique territory of blood supply and small area of lesion. The involvement of the ipsilateral corticospinal tract after the pyramidal decussation, or compression of the decussation, could cause the syndrome. We present a case of Opalski syndrome in a 71-year-old woman developed left hemiplegia and sudden difficulty for walking, accompanied by diplopia, headache, dysphagia and recurrent vomiting. Magnetic resonance image demonstrated a left lateral-medullary infarction. Angiography revealed distal and proximal stenosis of left vertebral artery and hypoplasia of right vertebral artery.

Humans , Lateral Medullary Syndrome , Paresis , Hemiplegia
Acta neurol. colomb ; 33(3): 182-187, jul.-set. 2017. tab, graf
Article in Spanish | LILACS | ID: biblio-886445


RESUMEN Se presenta un caso local de la variante de Opalski, del síndrome medular lateral (síndrome de Wallenberg) y se hace una revisión de la literatura sobre los síntomas de este cuadro clínico, así como las posibles razones que explican esta modificación en la expresión clínica. La variante de Opalski fue descrita en 1948, ochenta y nueve años después de la descripción original del síndrome por Adolf Wallenberg, su principal característica es la presencia de hemiparesia o hemiplejía del cuerpo del mismo lado de la lesión del bulbo raquídeo. La etiología es vascular en la mayoría de casos, por obstrucción de la arteria cerebelosa postero-inferior o de los ostium de vasos penetrantes que se originan en la arteria. No hay estudios que reporten la incidencia exacta de este cuadro clínico a nivel mundial, sin embargo, su presentación no es tan infrecuente. Lo más interesante al analizar cada caso, es que los hallazgos semiológicos son una muestra de la profunda organización del sistema nervioso.

SUMMARY A clinical case of the Opalski variant of the lateral medullary syndrome (Wallenberg's syndrome) is presented and a literature review about the symptoms of this clinical picture is done, as well as the possible reasons explaining this modification in the clinical expression. The Opalski variant was described in 1948, eighty-nine years after the original description of the syndrome by Adolf Wallenberg, its main feature is the presence of hemiparesis or hemiplegia of the ipsilateral side of the injury in the medulla oblongata. In most cases the etiology is vascular when the postero-inferior cerebellar artery is occluded or there is an obstruction of the ostium of penetrating vessels originating in the artery. There are no studies that accurately report the worldwide incidence of this clinical variant, however, its presentation is not uncommon. The most interesting discovery of each case is that semiological findings are a good example of the profound organization of the nervous system.

Vertebral Artery , Lateral Medullary Syndrome , Medulla Oblongata , Cerebral Infarction , Cranial Fossa, Posterior
Int. j. med. surg. sci. (Print) ; 4(1): 1141-1145, mar. 2017. ilus
Article in Spanish | LILACS | ID: biblio-1284372


El Síndrome de Wallenberg representa el 36% de los infartos del tronco cerebral. Se debe a la oclusión de la Arteria cerebelosa posterior inferior, afectando estructuras encontradas en el cerebelo y la parte lateral del bulbo raquídeo. El principal factor de riesgo es la aterosclerosis. El caso se trata de paciente masculino de 57 años de edad con antecedente de Infartos lacunares en cerebelo, Diabetes mellitus tipo 2No controlada, tabaquismo y dislipidemia. Inicia con cuadro clínico de cefalea occipital, intensa, súbita, sin atenuantes concomitantemente vértigo, hipo, nauseas, vómitos y parestesia de miembros inferiores. Al examen físico pulsos periféricos disminuidos, presenta Síndrome de Horner, hipo, hipoestesia en hemicara izquierda y hemicuerpo contralateral, con ataxia, dismetría y disdiadococinesia. Se realiza IRM con difusión con conclusión diagnostica: Imagen hiperintensa de morfología irregular en el contorno lateral izquierdo del bulbo raquídeo por restricción molecular, compatible con evento isquémico (Síndrome Wallenberg). Se indica tratamiento antitrombótico y terapia física. Paciente es evaluado 2 meses después mostrando amplia mejoría de su cuadro. Es un síndrome muy específico, cuya manifestación clínica depende de la región anatómica afectada y abstrae al clínico de otros diagnósticos.

Wallenberg's Syndrome represents 36 % of strokes in the brainstem. It is due to occlusionof the inferior cerebellar artery, affecting structures found in the cerebellum and the lateral part of themedulla oblongata. The main risk factor is atherosclerosis. The case is about a 57-year-old male patient witha history of lacunar infarcts in the cerebellum, type 2 diabetes mellitus, smoking and dyslipidemia. It beginswith an intense and sudden occipital headache, without attenuating, concomitantly vertigo, hiccups, nausea,vomiting and paresthesia of lower limbs. At the physical examination, decreased peripheral pulses presentHorner's syndrome, hypoesthesia, hypoesthesia in left hemiface, and contralateral hemibody, with ataxia,dysmetria and dysdiadochokinesia. MRI was performed with diffusion with diagnostic Hyperintense image ofirregular morphology in the left lateral contour of the medulla oblongata, compatible with ischemic event(Wallenberg syndrome). Antithrombotic treatment and physical therapy was indicated. Patient was evaluated2 months later showing ample improvement. It is a very specific syndrome, whose clinical manifestationdepends on the anatomical region affected and abstracts the clinician from other diagnoses.

Humans , Male , Middle Aged , Lateral Medullary Syndrome/diagnosis , Lateral Medullary Syndrome/therapy , Factor Xa Inhibitors/therapeutic use , Magnetic Resonance Imaging
Article in English | WPRIM | ID: wpr-51374


We studied three patients who developed left unilateral punctate keratitis after suffering left-sided Wallenberg Syndrome. A complex evolution occurred in two of them. In all cases, neurophysiological studies showed damage in the trigeminal sensory component at the bulbar level. Corneal involvement secondary to Wallenberg syndrome is a rare cause of unilateral superficial punctate keratitis. The loss of corneal sensitivity caused by trigeminal neuropathy leads to epithelial erosions that are frequently unobserved by the patient, resulting in a high risk of corneal-ulcer development with the possibility of superinfection. Neurophysiological studies can help to locate the anatomical level of damage at the ophthalmic branch of the trigeminal nerve, confirming the suspected etiology of stroke, and demonstrating that prior vascular involvement coincides with the location of trigeminal nerve damage. In some of these patients, oculofacial pain is a distinctive feature.

Aged , Cornea/pathology , Diagnosis, Differential , Female , Humans , Keratitis/diagnosis , Lateral Medullary Syndrome/complications , Middle Aged
Article in Korean | WPRIM | ID: wpr-11848


The Wallenberg's syndrome is produced by infarction of lateral medulla. Isolated ipsilateral axial lateropulsion without other common symptoms of Wallenberg syndrome has rarely been reported as manifestation of lateral medullary infarction. The responsible anatomical structure of ipsilateral axial lateropulsion is still uncertain. We describe a patient with lateral medullary infarction who present with isolated ipsilateral axial lateropulsion without other symptoms of Wallenberg syndrome.

Humans , Infarction , Lateral Medullary Syndrome
Acta méd. peru ; 30(2): 79-85, abr.-jun. 2013. ilus, graf, mapas, tab
Article in Spanish | LILACS, LIPECS | ID: lil-692315


Introducción: En agosto del 2009 operamos el primer paciente con esclerosis lateral amiotrófica (ELA). Sin embargo, hasta la fecha, todo los investigadores informan que no hay cura para esta enfermedad. Objetivo: Demostrar que la ELA puede ser detenida y mejorada mediante un transplante de epiplón. Material y Método: Presentamos a 13 pacientes con formas bulbar y espinal de ELA. Durante la cirugía encontramos: 1) variantes anatómicas del segmento V4 de las arterias vertebrales, 2) aterosclerosis moderada o severa en ambos segmentos V4, 3) algunas arterias circunflejas originadas desde las arterias espinales anteroventrales (AEAVs) exsangües, 4) hipotrofia de raicillas nerviosas en la hilera de los nervios IX, X y XI, 5) en algunos casos, hipotrofia de la superficie anterior de las pirámides e 6) hipotrofia de raíces anteriores en C5 - C6.Todos ellos recibieron transplante de epiplón a la superficie anterior, lateral y posterior de la medula oblongada y en 5 pacientes, un transplante adicional a nivel C5-C6. Resultados: La mejoría neurológica fue observada desde el primer día de la operación y fue mayor durante los primeros días o semanas de la cirugía que en los siguientes meses. Actualmente, 2 pacientes con 8 y 12 meses de evolución postoperatoria han mejorado en un 90% los síntomas de la forma bulbar de ELA. Conclusión: Estos resultados indican que la forma bulbar de ELA es originada por isquemia progresiva en el territorio intraparenquimatoso de las AEAVs y la forma espinal por isquemia en la arteria espinal anterior, pero a nivel C5 a T1. Eso explicaría porque su revascularización por medio del epiplón produjo mejoría neurológica.

Introduction: In August 2009 we performed surgery for the first time in a patient with amyotrophic lateral sclerosis (ALE). However, every published report about ALE mentions there is no cure for this condition. Objective: To prove that the progression of ALE may be stopped and the condition may be improved using an epiplon transplant. Material and Method: This report presents our experience with 13 ALE patients, affected by the bulbar and spinal forms of the disease. During the surgical procedures, we made the following findings: 1) anatomical variants in the V4 segment of vertebral arteries, 2) moderate or severe atherosclerosis in both V4 segments, 3) some circumflex blood vessels originating from the low-flow anterior and ventral spinal arteries, 4) hypothrophy of small nerve roots in the pathway of IX, X, and XI cranial nerves, 5) in some cases, hypothrophy of the anterior surface of the pyramids, and 6) hypothrophy of the anterior roots in C5 - C6. Every patient underwent an epiplon transplant upon the anterior, lateral, and posterior aspects of the medulla oblongata, and 5 patients underwent an additional transplant performed upon the C5-C6 territory. Results: Neurological improvement was seen from the first postoperative day, and it reached its maximum level during the first few days or weeks after surgery. Nowadays, 2 patients after eight and twelve months after surgery have had a 90% symptomatic improvement of the bulbar form of ALE. Conclusion: These results indicate that the bulbar form of ALE is caused by progressive ischemia in the parenchymal territory irrigated by the anterior and ventral spinal arteries, and that the spinal form is caused by ischemia in the anterior spinal artery, affecting the area between C5 and T1. This may explain why this revascularization procedure using epiplon tissue led to neurological improvement.

Humans , Male , Female , Adult , Middle Aged , Omentum/transplantation , Amyotrophic Lateral Sclerosis/surgery , Spinal Cord Ischemia , Lateral Medullary Syndrome , Pyramidal Tracts , Prospective Studies
Article in Korean | WPRIM | ID: wpr-761148


Sudden hearing loss and vertigo are the typical presentation of anterior inferior cerebellar artery infarction, but may rarely occur in posterior inferior cerebellar artery (PICA) infarction. Here we describe a 65-year-old man who presented with sudden hearing loss in his left ear and severe vertigo. The diffusion-weighted magnetic resonance imaging revealed acute infarction in the territory of PICA and cerebral angiography showed non-visualization of left vertebral artery. Sudden hearing loss and vertigo may be a presentation of PICA infarction.

Aged , Arteries , Cerebral Angiography , Ear , Hearing Loss, Sudden , Humans , Infarction , Lateral Medullary Syndrome , Magnetic Resonance Imaging , Pica , Vertebral Artery , Vertigo
West Indian med. j ; 61(7): 746-750, Oct. 2012. ilus, tab
Article in English | LILACS | ID: lil-672994


We describe two cases of lateral medullary syndrome at the University Hospital of the West Indies, Mona, Jamaica. This diagnosis is often missed and not well understood, so we will discuss the underlying pathophysiology.

Se describen dos casos de síndrome medular lateral en el Hospital Universitario de West Indies, Mona, Jamaica. Este diagnóstico pasa a menudo inadvertido y no es bien entendido. Por esa razón se discute aquí la patofisiología subyacente.

Humans , Male , Middle Aged , Lateral Medullary Syndrome/diagnosis , Jamaica , Lateral Medullary Syndrome/physiopathology , Magnetic Resonance Imaging
Article in Korean | WPRIM | ID: wpr-656329


Rotational vertebral artery syndrome (RVAS) is a rare disease entity. RVAS is characterized by recurrent attacks of vertigo, nystagmus, ataxia, and tinnitus, which is elicited by head-rotation when the contralateral dominant vertebral artery is compressed, leading to an acute reduction of blood flow in the vertebrobasilar territory. A 50-year-old female presented with a whirling type vertigo which, when turning her head to the left, compressed the dominant right vertebral artery, as documented by the dynamic neck angiography. The patient underwent posterior cervical decompression of the foramina transversaria and the surrounding soft tissues at the level of C1-C2. The patients's symptom disappeared after the surgery and she has been followed up for the last four months without the recurrence of vertigo.

Angiography , Ataxia , Decompression , Female , Head , Humans , Lateral Medullary Syndrome , Middle Aged , Neck , Rare Diseases , Recurrence , Tinnitus , Vertebral Artery , Vertigo
Article in Korean | WPRIM | ID: wpr-654506


The frequency of vertebral artery aneurysm is rare and a common presenting sign is subarachnoid hemorrhage. Lateral medullary syndrome is characterized by loss of pain and temperature sensation on the contra lateral lesion side of the body and ipsilateral lesion side of the face, dysphagia, dysarthria, ataxia, vertigo, nystagmus, and Horner syndrome. Vertebral artery dissecting aneurysm is a common cause of lateral medullary infarction. We present a rare case of a 46-year old male patient that developed ischemic attack presenting as transient lateral medullary syndrome due to thrombosed-fusiform aneurysm of vertebral artery. He was treated with aspirin and heparin, and then discharged with complete resolution of symptoms.

Aneurysm , Aneurysm, Dissecting , Aspirin , Ataxia , Deglutition Disorders , Dysarthria , Heparin , Horner Syndrome , Humans , Infarction , Lateral Medullary Syndrome , Male , Sensation , Subarachnoid Hemorrhage , Thrombosis , Vertebral Artery , Vertigo
Rev. arg. morfol ; 1(4)2012.
Article in Spanish | LILACS | ID: lil-733601


Introducción: Este trabajo describe la existenciade una variante anatómica: La hipoplasia de las arteriasvertebrales, definida como un estrechamiento de su diámetro luminal (< 2-3 mm) que conlleva una disminuciónde la velocidad del flujo sanguíneo encefálico. Material yMétodo: En nuestra experiencia se disecaron 20 encéfalos formalizados al 10% de la Cátedra de Anatomía de la Fac. de Cs. Médicas de la Universidad Nacional de Córdoba con técnica de disección clásica. Resultados: La arteria vertebral izquierda es dominante con respecto a la vertebral derecha y que emergen del segmento V4. Conclusión: La importancia del conocimiento anatómico de la arteria vertebral y sus variables facilita el diagnóstico precoz de la presencia de hipoplasia arterial, la cual se asocia a ciertos trastornosneurológicos, como son la migraña con aura, la isquemia cerebelar, (Sindrome de Wallemberg), disección de laarteria vertebral o cambios ateroescleróticos.

Introduction: This paper describes the existencean anatomical variant: hypoplasia of the arteriesvertebral defined as a narrowing of luminal diameter (<2-3 mm) which leads to a decreaseof cerebral blood flow velocity. Material andMethod. In our experience 20 brains formalized 10% of the Department of Anatomy, Faculty of Cs were dissected. Medical, National University of Cordoba with classical dissection technique. Results: The left vertebral artery is dominant with respect to the right vertebral V4 and emerging segment. Conclusion: The importance of anatomical knowledge of the vertebral artery and variables facilitates the early diagnosis of the presence of arterial hypoplasia, which is associated with certain disordersneurological disorders, such as migraine with aura, cerebellar ischemia (Wallenberg Syndrome), dissectionvertebral artery or atherosclerotic changes.

Humans , Male , Female , Vertebral Artery/pathology , Lateral Medullary Syndrome , Lateral Medullary Syndrome/pathology