Mobile phone radiation induced plasma protein alterations and eye pathology in newly born mice

The hazardous health effect of the exposure to 900-1800 MHz radiofrequency electromagnetic fields [RF-EMF] which emitted from mobile phones was investigated on the plasma protein and eye of newly born mice. Twenty one newly born mice were divided into 3 groups, the 1[st] group served as control, the 2[nd] group exposed to mobile phone radiation daily for one month [45 min/day] and the 3[rd] group remained one month following the end of exposure. The results showed deleterious changes in the plasma protein pattern by electrophoretic analysis. Also, the microscopic examination demonstrated numerous histopathological and histochemical changes in the eye mainly represented by degenerated, hemorrhagic areas and detachment in some layers of the eye with alteration in collagen, polysaccharides, total protein and marked increase in amyloid beta [beta] protein contents of newly born mice exposed to RF-EMF from mobile phone [45 min/day] for one month as well as after one month following the end of exposure. It was concluded that the exposure to mobile phone radiation causes plasma proteins alterations and eye pathology in newly born mice

Radiation emitted from mobile phone induces amyloidosis features in some tissues of infant mice

Investigating the effects of mobile phone-emitted radiation [MPR] on inducing histopathological changes associated with amyloidosis feature in liver, kidney and brain of infant mice. Twenty one infant mice [aged 1 day] were assigned to 3 groups, the 1[st] group served as control, the 2[nd] group exposed to mobile phone radiation [MPR] daily for one month [¾ h /day] and the 3[rd] group remained for one month after stopping radiation exposure. There were different degrees of damage related to amyloidosis feature in these organs subsequent to MPR exposure. One month post exposure there was an increase in the degree of damage related to amyloidosis feature. The results of this study showed that MPR leads to histopathological changes associated with amyloidosis feature in the liver, kidney and brain of infant mice

New metal complexes of semicarbazones derived from para phenylene diamine of NOO/or NO donor sites and their antimicrobial screening

The synthesis and characterization of new Mn[II]. Co[II] Ni[II] and Cu[II] complexes with semicarbazone ligands derived from para phenylene diamine [where L[1] = Semicarbazide-4-yl-benzene-4 [2-hydroxybenzalde-hyde semicarbazone] [SBFIBS], L[2] = Betizene-1, 4-bis-[2-hydroxybenz-aldehyde semicarbazone-4yl] [BBHBS], L[3] = Semicarbazide-4yl-benzene-4 [2-hydroxyacetophen-one-semicarbazone] SBHAS and L[4] = Semicarbazide-4yl-benzene-4 [2-nitrobenzaldehyde semicarbazone] [SBNBS]] are reported. The ligands contain NOO/or NO donor sites. The elemental analysis suggests different stoichiometries 1: 2: 1 and 1: 2 [M: L]. IR spectra data indicate covalent bond through the oxygen atom of the hydroxyl group, coordination of the carbonyl oxygen and the azomethine nitrogen / or nitrogen of the amine group to the metal ion. TGA determined whether the water or solvent molecules are inside or outside the coordination sphere. Magnetic susceptibility and electronic data are in favour of octahedral structures except in the case of Co [II] complexes of ligand BBHBS and SBHAS as they favour the tetrahedral structure. The biological activity of the complexes has been tested. These complexes showed promising antifungal activities

Platelet aggregation in cerebrovascular ischemic stroke

The present study was performed on 15 patients [8 patients with acute stroke within 48 hours from the attacks and 7 patients with chronic stroke after 6 months from the attack] and 10 healthy normal control subjects. The patients and controls were subjected to ADP, collagen and ristocetin included aggregation. Analysis of aggregation revealed there was a statistically significant elevation on using ADP agonists in acute stroke group compared to the control group. Also, there was a statistically significant elevation on using ADP and collagen agonists in acute stroke group compared to the chronic stroke group. The results of this study suggested that, platelet hyperaggregability was observed soon after the acute event, but the test returned progressively to normal. So, there was lack of hyperaggregability to the chronic stage

Action of grignard reagents on halogenated phthalimides

3-BENZALPHTHAL1MIDINES [VI] and [X] were obtained by interaction of benzylmagnesium chloride with corresponding phthalimides then dehydration of the resultant 3-benzyl-3-hydroxy-phthalimidines [IV] and [VIII]. Interaction of phenylmagnesium bromide and ethylrnagnesium iodide with brornoph thalimides [I] is also discussed