A Case of Bilateral Sudden Hearing Loss and Tinnitus after Salicylate Intoxication

Salicylate, the active ingredient of aspirin can cause sensorineural hearing loss and tinnitus when plasma concentrations reach a critical level. The ototoxic mechanisms of salicylate remain unclear but hearing and tinnitus usually recovers a few days after intoxication. There have been few reports of salicylate-induced ototoxicity in Korea, and the majority is caused by a low dose of aspirin. Herein, we report a case of sudden hearing loss and tinnitus after acute salicylate intoxication and review recent updates on salicylate ototoxicity.

A Case of Schwannoma of the Nasal Tip: Resection Using an External Rhinoplasty Approach / 대한이비인후과학회지

Schwannoma is a benign tumor originating from the peripheral nerve sheath. A 16-year-old man have experienced progressive swelling of the nasal tip over 4 years. Computed tomography scan showed a 2.5 cm sized soft tissue density at the nasal tip. It distorted and dislocated the lower lateral cartilage. The mass was resected using an external rhinoplasty approach. The nasal tip was reconstructed by tip surgery and the soft tissue defect under the skin due to mass was filled with autologous dermal graft. The pathologic diagnosis was a schwannoma. A nasal tip schwannoma is extremely rare. Here, we present and discuss the clinical and surgical aspects of schwannoma arising on the nasal tip.

A Case of Solitary Neurofibroma of the External Auditory Canal / 대한이비인후과학회지

Neurofibroma is a relatively common neurogenic tumor originating from schwann cells or fibroblasts in peripheral nerve sheaths. It is classified as solitary or multiple. Solitary lesions are not usually associated with neurofibromatosis or von Recklinghausen's disease. We report a case of solitary neurofibroma occurred in the right external auditory canal (EAC) in a 30-year-old male who complained of progressive hearing loss and earfullness for 5 years. This diffuse painless mass displaced the auricle laterally and obstructed EAC completely. The lesion was partially excised by retroauricular approach and diagnosed as neurofibroma. This patient does not have any other evidences of neurofibromatosis. A complete excision in this case was not mandatory as malignant change is rare; moreover, it would require extensive resection of periauricular skin, subcutaneous tissue, cartilaginous ear canal and temporalis muscle, followed by reconstruction. We present the clinical characteristics and therapeutic methods of a solitary neurofibroma originating from EAC.