RESUMEN
Sudden sensorineural hearing loss (SNHL) due to blunt head trauma is a rare condition. Possible causes of SNHL by head trauma include cellular injury, perilymphatic fistula, labyrinthine concussion due to microfracture in inner ear and blunt trauma with simultaneous acute labyrithitis. We report here on two unusual cases in which cochleovestibular functions were totally damaged in patients with sudden SNHL due to blunt head trauma. Both cases presented with vertigo, tinnitus and a sudden onset of hearing impairment in the unilateral ear after blunt trauma. Audiograms revealed a profound to severe unilateral SNHL. Caloric testing revealed a decreased caloric response of 100% in the ipsilateral side compared to the contralateral side. Vestibular evoked myogenic potential testing revealed no response in the ipsilateral ear. Neither patient recovered hearing despite oral and/or intratympanic steroid therapy.
Asunto(s)
Humanos , Pruebas Calóricas , Traumatismos Craneocerebrales , Oído , Oído Interno , Fístula , Cabeza , Traumatismos Cerrados de la Cabeza , Audición , Pérdida Auditiva , Pérdida Auditiva Sensorineural , Pérdida Auditiva Súbita , Laberintitis , Acúfeno , VértigoRESUMEN
Sudden sensorineural hearing loss (SNHL) due to blunt head trauma is a rare condition. Possible causes of SNHL by head trauma include cellular injury, perilymphatic fistula, labyrinthine concussion due to microfracture in inner ear and blunt trauma with simultaneous acute labyrithitis. We report here on two unusual cases in which cochleovestibular functions were totally damaged in patients with sudden SNHL due to blunt head trauma. Both cases presented with vertigo, tinnitus and a sudden onset of hearing impairment in the unilateral ear after blunt trauma. Audiograms revealed a profound to severe unilateral SNHL. Caloric testing revealed a decreased caloric response of 100% in the ipsilateral side compared to the contralateral side. Vestibular evoked myogenic potential testing revealed no response in the ipsilateral ear. Neither patient recovered hearing despite oral and/or intratympanic steroid therapy.
Asunto(s)
Humanos , Pruebas Calóricas , Traumatismos Craneocerebrales , Oído , Oído Interno , Fístula , Cabeza , Traumatismos Cerrados de la Cabeza , Audición , Pérdida Auditiva , Pérdida Auditiva Sensorineural , Pérdida Auditiva Súbita , Laberintitis , Acúfeno , VértigoRESUMEN
@# ObjectiveTo study memory impairment of closed head injured(CHI) patients during later recovering period. MethodsMemory function was examined in 20 CHI patients and 20 matched healthy controls by Clinical Memory Scale(CMS). ResultsThe scores of 5 sub tests and MQ of CHI group were significant lower than that of control group, the rate of MQ ≤ 79 in CHI patients was significant higher than in controls. Conclusion There are severe memory impairment in CHI patients during later period, the impairment of recall is more serious than that of recognition.
RESUMEN
We reported our experience of monitoring head-injury patients by means of TCD. The onset, duration, and amplitude of the altered flow velocities were somewhat different from those that are seen with the blood flow velocities after aneurysmal subarachnoid hemorrhage. Twenty patients suffering closed head injury were studied using TCD recording of the middle cerebral artery. On admission, twenty patients had Glasgow coma score of 10 or less. Serial examinations were began within 24 hours of trauma and continued until the patient was discharged. The increase of blood flow velocity began as early as 24 hours after injury, reached a maximum around Day 7 and lasted until 3 weeks after injury. The technique was simple, non-invasive, and repeatable so that diagnostic assessments can be made and the patient can be followed for therapeutic efficacy.
Asunto(s)
Humanos , Velocidad del Flujo Sanguíneo , Coma , Traumatismos Cerrados de la Cabeza , Arteria Cerebral Media , Hemorragia SubaracnoideaRESUMEN
The traumatic intracranial aneurysm within the posterior cranial fossa is extremely rare. The case of a 18-year-old boy who developed an saccular aneurysm lately in the posterior fossa after a blunt head injury is reported. Repeated follow-up angiography demonstrated an saccular aneurysm at the junction of vertebral artery and posterior inferior cerebellar artery. Early brain MRI disclosed neither aneurysm nor mass. Late brain MRI revealed a partially thrombosed saccular aneurysm at the cerebello pontine angle. Follow-up MRI and angiography is recommended if traumatic aneurysm is suspected.
Asunto(s)
Adolescente , Humanos , Masculino , Aneurisma , Angiografía , Arterias , Encéfalo , Fosa Craneal Posterior , Estudios de Seguimiento , Traumatismos Cerrados de la Cabeza , Aneurisma Intracraneal , Imagen por Resonancia Magnética , Arteria VertebralRESUMEN
Two cases of the traumatic aneurysms of the pericallosal artery that occurred after closed head injury are reported. The mental deterioration, urinary in continence and crural-predominent hemiparesis are attributable to the delayed intracranial hemorrhage after rupture of the traumatic aneurysms of the pericallosal artery. As soon as the diagnosis has been confirmed by the cerebral angiography, surgical treatment is recommended to reduce the high mortality and morbidity and the procedure as in the congenital aneurysm.
Asunto(s)
Aneurisma , Arterias , Angiografía Cerebral , Diagnóstico , Traumatismos Cerrados de la Cabeza , Hemorragias Intracraneales , Mortalidad , Paresia , RoturaRESUMEN
A few delayed epistaxis was reported, and it is an unusual complication of closed head injury. We experienced 2 cases of delayed epistaxis and monoocular blindness by cavernous vascular lesions(traumatic aneurysm and C. C. F.). We confirmed the vascular lesions by carotid angiography and Brain C. T. They were treated by carotid ligation and carotid ligation with embolization, and we mer with good results. We present 2 cases of the delayed epistaxis complicated by traumatic lesions of cavernous internal carotid artery, and also the literatures were reviewed.