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Objective To explore the feasibility of using short-latency somatosensory evoked potentials (SLSEP) to quantitate Deqi.Methods A randomized crossover controlled trial was carried out. Healthy subjects were enrolled and allocated to treatment (thick needle, deep insertion and manipulation for Deqi) and control (thin needle, shallow insertion and no manipulation without Deqi) groups. Somatosensory evoked potentials were recorded before, during and after acupuncture. Deqi was assessed using the score scale in the subjets. The effects of Deqi and no Deqi at point Sanyinjiao (SP 6) on the potentials were observed.Results The preliminary exploration of the feasibility by the trial test showed that the effect of Deqi on short-latency somatosensory evoked potentials had certain regularity. It was worthy to be observed.Conclusion The plan is feasible. The formal test can be conducted.
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@#ObjectiveTo evaluate central nervous system function of patients with coronary cardiac diease by short latency somatosensory evoked potentials (SSEP).MethodsThe cerebral and spinal somatosensory evoked potentials were recorded by stimulating median nerve in 43 patients with coronary cardiac disease but without apparent nervous symptoms and 14 healthy control subjects.ResultsThe lactency periods and central conductive time of N13, N20 and P25 wave were significantly prolonged in patients with myocardial infarction (MI) or angina pectoris (AP) when compared with normal controls (P<0.05～0.001). The lactency periods and central conductive time of N20 and P25 wave recorded in MI patients were longer than those recorded in AP patients (P<0.01～0.001).ConclusionThe subclinical nervous damages in the central somatosensory pathway from spinal cord to cerebral cortex is present in patients with coronary cardiac disease especially myocardial infarction.
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Median, ulnar and tibial short-latency somatosensory-evoked potentials were recorded from 112 neurologically normal subjects. Absolute latencies and interpeak latencies were analysed. Absolute latencies were correlated with height (r=0.37-0.59, P<0.01) but not with age (r=0.16-0.55, P>0.05). Interpeak latencies were not correlated either with height or age, except median/ulnar N9-N19 which were correlated with age and height (r=0.34, .044, P<0.01). Males had longer absolute and interpeak latencies than females (P<0.05) which were probably due to height difference (P<0.001). Hence, height is the most important variables for predicting absolute latencies, except in age group over 60 year old that the absolute and interpeak latencies were significantly delayed (P<0.05). The equations foe such predictions were verified. Hence, any absolute latencies should be corrected for height prior to considering them to be abnormal.
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Short latency somatosensory evoked potentials (SSEPs) were elicited by stimulation of the median and tibial nerves in 55 patients with cerebrovascular diseases. There were 28 males and 27 females. The mean age was 52.7+14.9 years (range 20-83). Every patient presented as an acute hemiplegic with and without sensory loss. The CT findings were classified into 6 groups, thalamic, internal capsule, subcortical white matter, basal ganglia, occlusion of proximal branch of middle cerebral artery and transient ischemic attack. The abnormality rate of median SSEPs was 63.6 percent (35/55) and tibial SSEPs was 67.3 percent (37/55). In thalamic stroke both median and tibial SSEPs abnormality rates were 80 percent (8/10), in ganglionic stroke the rates were 75 percent (9/12). The internal capsule abnormality rates were 75 percent (9/15). Subcortical white matter there was 57.1 percent (4/7) and 80 percent (8/10) in occlusion of the main branch of middle cerebral artery. The SSEPs specificity, positive and negative predictive values between cerebral hemorrhage and infarction were 42.8, 47.4 and 87.2 percent respectively. Hence, SSEPs might be of value in localizing anatomical lesions but not to differentiate between hemorrhagic and ischemic stroke.