Résumé
Objective:To observe the clinical effects of electroacupuncture (EA) plus acuoint-injection for nerve root sciatica. Methods:A total of 114 cases were randomly divided into an observation group and a control group, 57 cases in each group. The observation group was given EA plus acupoint-injection therapy with Cobamamide. The control group was given oral administration of Diclofenac Sodium Sustained-release Tablets plus muscular injection of Cobamamide. After one-course treatment, the clinical effects were compared between the two groups. Results:The total effective rate was 96.5%in the observation group and 91.2%in the control group, without a statistical significance in the difference between the two groups (P>0.05). The curative rate was 57.9%in the observation group and 21.1% in the control group, with a statistical significance in the difference between the two groups (P Conclusion: EA plus acupoint-injection therapy for sciatica of nerve roots is remarkable in the clinical effect and needs promotion in clinic.
Résumé
Objective To observe the effect of electroacupuncture (EA) on the expression of NF-?B in cerebral ischemia area of cerebral ischemia-reperfusion rats and the neuron apoptosis in cortex by EA on shuigou and neiguan (left) point, and study the protective effect and its mechanism of EA on the cerebral ischemia. Method Cerebral ischemia reperfusion model was established by occlusion of the middle cerebral artery for 30 min and then removal, then EA shuigou and neiguan left points of experimental rats. Neuronal apoptosis in cortex was observed by the terminal deoxynucleotidyl transferase-mediated dUTP-biotin nick end labeling (TUNEL), NF-?B protein in the ischemia cortex was detected by immunohistochemistry. Result The neuronal apoptosis and the expression of NF-?B protein in the ischemia cortex was deteded of ischemia side of EA treatment groups were lower comparing with model group. Conclusion EA can lighten the neurons injury after cerebral ischemia-reperfusion by decreasing the expression of NF-?B protein and the neuronal apoptosis in the ischemia cortex.