Résumé
Introduction: In developing nations, the increased prevalence of type 2 diabetes mellitus (T2DM) has resulted in significant morbidity and socioeconomic consequences. Hypomagnesemia has been associated with insulin resistance and related micro- vascular consequences due to its ability to trigger hyperglycemia. Although many research articles on glycemic control have been published in recent years, the latest therapeutic approaches may not be feasible to all. As a result, prioritising preventative and primary care research becomes critical. Aim and objective: The study is aimed at estimating serum magnesium concentration and glycated hemoglobin (HbA1c) levels in patients with Type 2 Diabetes mellitus. This will help us evaluate how glycemic control in Diabetes can influence serum Magnesium levels. Method: The study consists of 50 consenting patients who came for routine blood investigations. Those patients having high random blood sugar were selected and divided into two groups based on their age. i.e. 25-50, 51-75. Other parameters con- sidered were age, gender and diet. After an overnight fasting, blood of the consenting patient was collected. Magnesium was measured by Cobas 6000 using the calorimeter end-point method. The other blood sample was transferred into an EDTA test tube which was used to estimate HbA1c level. Result: Participants were divided into two groups based on their age. i.e. 25-50, 51-75. The mean serum magnesium levels were 1.46 mg/dL for group 1 and 1.3 mg/dL for group 2. The mean HbA1c levels for group 1 were 7.65 and and 8.36 for group 2 respectively. According to Pearson’s correlation coefficient, inverse correlation was found between HbA1c levels and Serum magnesium levels. Conclusion: Magnesium insufficiency has been linked to a higher incidence of diabetic due to poor glycemic management in people with diabetes. To avoid such problems and maintain glycemic control, dietary supplements may be recommended. Large-scale clinical research is also required.
Résumé
Introduction: Epidermoid cysts are cystic malformations filled with keratin and lipid rich debris. They generally present as benign, soft, and freely movable, slowly enlarging, and non-tender masses, commonly located on the face, neck and trunk of the body. They are more commonly seen in ovaries and gonads, and less often in head and neck region. Approximately 7% of epidermoid cysts are present in the head and neck region and about 1.6% are found in the oral cavity. They can be either congenital or acquired in origin. Congenital epidermoid cyst occurs at third and fourth intra-uterine life due to entrapment of ectodermal elements entrapped during midline fusion of first and second branchial arches. However, acquired epidermal cysts occur due to implantation of epidermal elements following cystic transformation. Case presentation: In this article, we discuss a case of epidermoid cysts presenting in multiple areas of the face which clinically appeared to as acquired nevi (mole). Management/Prognosis: Based on clinical appearance and provisional diagnosis, treatment of mole was made. The lesions were excised by electrocautery and sent for histopathological evaluation. Conclusion: There was a drastic difference between the clinical appearance and histopathological picture seen. A thorough knowledge about etiology, clinical history and histopathology is needed to reach an accurate diagnosis
Résumé
Dentigerous cyst is the most common developmental odontogenic cyst, frequently noted as an incidental finding on radiographs. It commonly affects the permanent mandibular third molars and maxillary canines. We present a case of a dentigerous cyst involving an impacted mandibular second molar in a 21-year-old female.
Résumé
The keratocystic odontogenic tumor (KCOT), formerly known as odontogenic keratocyst, is a benign developmental odontogenic tumor with many distinguishing clinical and histologic features. Hard tissue deposits, which usually take the form of dystrophic calcifications, cartilaginous tissue, or dentinoid, are uncommon findings in the connective tissue capsule of the KCOT. We report a case of a 33‑year‑old female with KCOT showing osseous tissue and calcified deposits close to its epithelial lining, which is an extremely rare occurrence. A brief review on the reported prevalence of hard tissue deposits in KCOTs and possible mechanisms that has been implicated in mineralization and bone formation has been discussed.