Noncardiogenic Pulmonary Edema Related to Airway Obstruction / 대한마취과학회지

Pulmonary edema is a recognized comphcation of acute airway obstruction. When pulmonary edema occurs, it usually follows relief of obstruction and is likely to be of noncardiogenic origin. We present a case of noncanhogenic pulmonary edema that occured in a ncy woman who was transfered to our hospital, because of endotracheal intubation failure and unrelieved bronchospasm, during general anesthesia for Cesarian section.

A Management System of Data for Anesthesia Department and Patients Using the Personal Computer / 대한마취과학회지

To build a clinical data base for the patient is one of the important works in the department of anesthesiology. Many of the works can be accomplished by presonal computer, of which include registration of patient data, searching for articles, and analysis of clinical data etc. We would like to introduce a computer programs for the management of various data of anesthesia department and patients. We expect that more advanced and beneficial programs appear to the management of data for anesthesia department and patients.

Effect of Labetalol on Cardiovascular Responses to Laryngoscopy and Endotracheal Intubation / 대한마취과학회지

Arterial blood pressure, pulse rate and rate pressure product changes following tracheal intubation were studied in 50 patients undergoing elective surgical procedures who received a thiopental-succinylcholine anesthetic intubation sequence. Three treatment groups and a control group were observed. Intravenous labetalol doses of 0.25 mg/kg, 0.5 mg/kg and 1.0 mg/kg, injected prior to anesthesia, were compared with respect to their effect on the cardiovascular sequences to direct laryngoscopy followed by the passage of an endotraeheal tube. The increases in heart rate and rate-pressure product associated with tracheal intubation were significantly prevented in labetalol treated patients, significantly. The increase in arterial pressure was prevented, insignificantly. However, it was dose-dependent. From the above result, a pre-induction dose of labetalol was effective in attenuating the pressure response to laryngoscopy and intubation.