Résumé
Acute pancreatitis with diaphragmatic hernia is rare, and furthermore, very few were reported in the case without incarceration. The suggested mechanism of acute pancreatitis with diaphragmatic hernia is that herniation of pancreas itself. The other possible mechanism is pancreatic ischemia due to traction after aggravation of hernia or acute distention of stomach. We report a case of acute pancreatitis due to diaphragmatic hernia with no evidence of herniation of pancreas. A 78-year-old male was administered for epigastric pain and dyspnea. The radiologic and laboratory result demonstrated an acute pancreatitis with diaphragmatic hernia without any evidence of herniation of pancreas. The patient was managed conservatively to reduce the hernia and to treat pancreatitis.
Sujets)
Adulte , Sujet âgé , Humains , Mâle , Dyspnée , Hernie , Hernie diaphragmatique , Ischémie , Pancréas , Pancréatite , Estomac , TractionRésumé
Plummer-Vinson syndrome is characterized by dysphagia, iron deficiency anemia, and upper esophageal web. The associated symptoms can be resolved by administering iron supplements as well as by endoscopic intervention. Relapse in patients with Plummer-Vinson syndrome is very rare. We describe a case of a 42-year-old woman with Plummer-Vinson syndrome whose symptoms were successfully treated with endoscopic dilatation and iron supplementation at first admission; however, 1 year later, she revisited our hospital because of dysphagia. On second admission, investigations revealed esophageal web relapse in Plummer-Vinson syndrome. She was again successfully treated with endoscopic dilatation and iron supplementation. After first admission, her anemia was not normalized due to poor compliance and loss of follow-up. We experienced a case of esophageal web relapse due to uncorrected iron deficiency anemia in a patient with Plummer-Vinson syndrome. This experience indicates that continuous iron supplementation and long-term follow-up is important in patients with Plummer-Vinson syndrome.
Sujets)
Adulte , Femelle , Humains , Anémie , Anémie par carence en fer , Compliance , Troubles de la déglutition , Dilatation , Études de suivi , Fer , Syndrome de Plummer-Vinson , RécidiveRésumé
Plummer-Vinson syndrome is characterized by dysphagia, iron deficiency anemia, and upper esophageal web. The associated symptoms can be resolved by administering iron supplements as well as by endoscopic intervention. Relapse in patients with Plummer-Vinson syndrome is very rare. We describe a case of a 42-year-old woman with Plummer-Vinson syndrome whose symptoms were successfully treated with endoscopic dilatation and iron supplementation at first admission; however, 1 year later, she revisited our hospital because of dysphagia. On second admission, investigations revealed esophageal web relapse in Plummer-Vinson syndrome. She was again successfully treated with endoscopic dilatation and iron supplementation. After first admission, her anemia was not normalized due to poor compliance and loss of follow-up. We experienced a case of esophageal web relapse due to uncorrected iron deficiency anemia in a patient with Plummer-Vinson syndrome. This experience indicates that continuous iron supplementation and long-term follow-up is important in patients with Plummer-Vinson syndrome.