[Effects of adentonsillectomy on pulmonary function parameters]

Adenotonsillar hypertrophy is a common finding among children which in untreated cases may result in serious cardiopulmonary problems. There are limited studies about the effect of adenotonsillectomy on pulmonary function parameters. Does adenotonsillectomy affect on pulmonary function? This study is performed to reveal this purpose. In a pre- and post-treatment clinical trial on 39 children between 6-13 years old with indication of adenotonsillectomy due to obstructive respiratory symptoms [because of prominent hypertrophy of tonsils and/or adenoid tissue], 5 main Spirometric parameters weere measured before and 2 months after adenotonsillectomy with a standard method and data were analyzed by paired t-test. 39 children [56% male and 44% female] with a mean age of 8.95 +/- 1.93 were evaluated. 4 main Spirometric parameters increased: FEV1/FVC from 89.H12.7 to 97 +/- 4.6, FEV0.5/FVC from 70.9 +/- 15.5 to 81.U11.8, FEV25-75 from 1.9 +/- 0.6 to 2.2 +/- 0.7, MEF50 from 2 +/- 0.6 to 2.4 +/- 0.7 and all the improvement was statistically significant [P<0.0001]. There was no statistically significant change in FEV1/ PEFR index. Due to obvious changes of 4 main Spirometric parameters, it can be deduced that adenotonsillectomy is of effective roles in improvement of pulmonary function

[Effects of adenotonsillectomy on pulmonary arterial pressure]

There are several studies which have shown that adenotonsillar hypertrophy can cause airway obstruction, chronic alveolar hypoventilation and even may lead to severe cardiopulmonary complications like pulmonary artery hypertension. The aim of this study was to evaluate the effects of adenotonsillectomy on pulmonary arterial pressure in children with hypertrophic tonsils and adenoids. fourty children [21 males, 19 females] aged between 4 and 14 [mean 7/9 +/- 2/3] years with a diagnosis of upper airway obstruction due to hypetrophied tonsils and adenoids were included in our study. Pulmonary arterial pressure was measured by Doppler echocardiography both preoperatively, and 6 weeks after operation. Preoperative pulmonary arterial pressure [PAP] was 26/1 +/- 3/9 mmHg and postoperative PAP was 25/9 +/- 5 mmHg. Although in two cases out of three ones who had raised preoperative PAP, adenotonsillectomy resulted in decreasing the pressure, but there was no statistically significant difference between pre-operative and post-operative echocardiographic findings. It seems that whilst increasing of pulmomary arterial pressure [owing to Adenotonsillar hypertrophy], adenotonsillectomy can be of beneficial outcomes in decreasing this pressure