RESUMO
The characteristic features of Churg-Strauss syndrome are vasculitis, both intra- and extravascular granuloma formation with eosinophilic tissue infiltration, and strong association with asthma and peripheral eosinophilia. Churg-Strauss syndrome with pleural effusion is relatively common and only a few cases have previously been reported in Korea. We report a case of Churg-Strauss syndrome with pleural effusion confirmed by VATS.
Assuntos
Asma , Síndrome de Churg-Strauss , Eosinofilia , Eosinófilos , Granuloma , Coreia (Geográfico) , Derrame Pleural , Cirurgia Torácica Vídeoassistida , VasculiteRESUMO
Behcet's disease characterized by recurrent oral and genital ulceration and ocular inflammation has been recognized as a multi-system disorder with numerous manifestations including gastrointestinal tract. Terminal ileum and cecum are the most frequently involved portions of the gastrointestinal tract in Behcet's disease and esophageal involvement is very uncommon. We experienced a case of Behcet's disease involved esophageal and ileocecal region which had been diagnosed by endoscopy. A 20-year-old male was admitted by dysphagia and substernal discomfort. There was a history of recurrent aphthous stomatitis and genital ulcers. The large geographic ulcers surrounded with edematous mucosa were found at mid esophagus and the huge ulcer with exudate was noted on ileocecal valve area in endoscopic examination. The microscopic finding of endoscopic biopsy showed chronic nonspecific inflammation with necrotic inflammatory exudate. Pathergy test was positive. So he was diagnosed as Behcet's disease, and his symptoms and signs were resolved after steroid treatment.
Assuntos
Humanos , Masculino , Adulto Jovem , Biópsia , Ceco , Transtornos de Deglutição , Endoscopia , Esôfago , Exsudatos e Transudatos , Trato Gastrointestinal , Valva Ileocecal , Íleo , Inflamação , Mucosa , Estomatite Aftosa , ÚlceraRESUMO
Acute bilateral reexpansion pulmonary edema after pleurocentesis is a rare complication. In one case, bilateral reexpansion pulmonary edema after unilateral pleurocentensis in sarcoma was reported. Verious hypotheses regarding the mechanism of reexpansion pulmonary edema include increased capillary permeability due to hypoxic injury, decreased surfactant production, altered pulmonary perfusion and mechanical stretching of the membranes. Ragozzino et al suggested that the mechanism leading to unilateral reexpansion pulmonary edema involves the opposite lung when there is significant contralateral lung compression. Here we report a case of bilateral reexpansion pulmonary edema and acute respiratory distress syndrome after a unilateral pleurocentesis of a large pleural effusion with contralateral lung compression and increased interstitial lung marking underlying chronic liver disease.