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The Korean Journal of Critical Care Medicine ; : 286-289, 2012.
Artigo em Coreano | WPRIM | ID: wpr-651248

RESUMO

Brain death results in adverse pathophysiologic effects in many brain-dead donors with cardiovascular instability. We experienced a brain-dead donor with continuous renal replacement therapy (CRRT) who was in a severe metabolic, electrolyte derangement and poor pulmonary function. The thirty-nine-year-old male patient with subarachnoid hemorrhage and intraventricular hemorrhage was admitted into the intensive care unit (ICU). After sudden cardiac arrest, he went into a coma state and was referred to as a potential organ donor. When he was transferred, his vital sign was unstable even under the high dose of inotropics and vasopressors. Even with aggressive treatment, the level of blood sugar was 454 mg/dl, serum K+ 7.1 mEq/L, lactate 5.33 mmol/L and PaO2/FiO2 60.3. We decided to start CRRT with the mode of continuous venovenous hemodiafiltration (CVVHDF). After 12 hours of CRRT, vital sign was maintained well without vasopressors, and blood sugar, serum potassium and lactate levels returned to 195 of PaO2/FiO2. Therefore, he was able to donate his two kidneys and his liver.


Assuntos
Humanos , Masculino , Glicemia , Encéfalo , Morte Encefálica , Coma , Morte Súbita Cardíaca , Hemodiafiltração , Hemorragia , Unidades de Terapia Intensiva , Rim , Ácido Láctico , Fígado , Potássio , Terapia de Substituição Renal , Hemorragia Subaracnóidea , Doadores de Tecidos , Sinais Vitais
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