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Journal of Dental Anesthesia and Pain Medicine ; : 77-82, 2019.
Artigo em Inglês | WPRIM | ID: wpr-740007

RESUMO

It is well known that trigeminal nerve injury causes hyperexcitability in trigeminal ganglion neurons, which become sensitized. Long after trigeminal nerve damage, trigeminal spinal subnucleus caudalis and upper cervical spinal cord (C1/C2) nociceptive neurons become hyperactive and are sensitized, resulting in persistent orofacial pain. Communication between neurons and non-neuronal cells is believed to be involved in these mechanisms. In this article, the authors highlight several lines of evidence that neuron-glial cell and neuron macrophage communication have essential roles in persistent orofacial pain mechanisms associated with trigeminal nerve injury and/or orofacial inflammation.


Assuntos
Comunicação Celular , Medula Cervical , Dor Facial , Inflamação , Macrófagos , Neurônios , Nociceptores , Gânglio Trigeminal , Nervo Trigêmeo , Traumatismos do Nervo Trigêmeo , Núcleo Espinal do Trigêmeo
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