RESUMO
Congenital ocular motor apraxia is characterized by impaired voluntary saccades and abnormal head thrusts to induce a fixation. We report a case of a 7-year-old boy who shows typical finding of congenital ocular motor apraxia. He had a history of spasmus nutans. His developmental milestones were delayed. Brain MRI demonstrated cerebellar vermian hypoplasia, especially in the inferior portion. We report on a case of congenital ocular motor apraxia associated with cerebellar vermian hypoplasia.
Assuntos
Criança , Humanos , Lactente , Recém-Nascido , Masculino , Apraxias , Encéfalo , Cabeça , Imageamento por Ressonância Magnética , Movimentos Sacádicos , Espasmos InfantisRESUMO
Congenital ocular motor apraxia is characterized by impaired voluntary saccades and abnormal head thrusts to induce a fixation. We report a case of a 7-year-old boy who shows typical finding of congenital ocular motor apraxia. He had a history of spasmus nutans. His developmental milestones were delayed. Brain MRI demonstrated cerebellar vermian hypoplasia, especially in the inferior portion. We report on a case of congenital ocular motor apraxia associated with cerebellar vermian hypoplasia.
Assuntos
Criança , Humanos , Lactente , Recém-Nascido , Masculino , Apraxias , Encéfalo , Cabeça , Imageamento por Ressonância Magnética , Movimentos Sacádicos , Espasmos InfantisRESUMO
Papilledema, pupillary abnormalities, and nystagmus are common neuro-ophthalmologic signs in neurocysticercosis (NCC). Oculomotor palsy rarely occurs and usually accompanies compression of the midbrain by supratentorial or subarachonoid lesions with or without inflammation and hydrocephalus. Oculomotor palsy from NCC involving the midbrain parenchyme has rarely been described. We report on a patient who presented with oculomotor palsy caused by mesencephalic NCC. The patient showed recurrences of symptoms in association with steroid tapering.