Neurogenic stunned myocardium following hemorrhagic cerebral contusion

Neurogenic stunned myocardium NSM is a well-known complication of subarachnoidal hemorrhage, but has been reported rarely in association with other central nervous system disorders. A case of NSM is described in a patient with hemorrhagic brain contusion associated with cerebral edema. An 18-year-old man was admitted with severe cranial trauma following a car roll-over. Six days after admission, he developed findings suggestive for NSM. The troponin T and creatine kinase-MB level were elevated and echocardiogram showed apical and inferoposterior hypokinesis and diffuse left ventricular akinesis with severely reduced ejection fraction 18%. Invasive measurements confirmed low cardiac output. His cardiac function resolved completely within 6 days after decompressive craniotomy. This case supports the presumed unifying role of the increased intracranial pressure, probably triggering a vigorous sympathetic outflow hyperactivity leading to NSM

Necrotizing fasciitis caused by aeromonas sobria: a case report

Aeromonas sobria is facultative, oxidase-positive, anaerobic, flagellated gram-negative bacilli. Found commonly in sewage, soil, and blackish or fresh water. Commonly causing various diseases in immunocompromised patient, but in healthy patients causing diarrhea and soft tissue infections. We are reporting a case of incomplete septic abortion and necrotizing fasciitis caused by Aeromonas sobria, which was successfully diagnosed early and managed by aggressive surgical and medical managements

Neurogenic stunned myocardium following hemorrhagic cerebral contusion

Neurogenic stunned myocardium [NSM] is a well-known complication of subarachnoidal hemorrhage, but has been reported rarely in association with other central nervous system disorders. A case of NSM is described in a patient with hemorrhagic brain contusion associated with cerebral edema. An 18-year-old man was admitted with severe cranial trauma following a car roll-over. Six days after admission, he developed findings suggestive for NSM. The troponin T and creatine kinase-MB level were elevated and echocardiogram showed apical and inferoposterior hypokinesis and diffuse left ventricular akinesis with severely reduced ejection fraction [18%]. Invasive measurements confirmed low cardiac output. His cardiac function resolved completely within 6 days after decompressive craniotomy. This case supports the presumed unifying role of the increased intracranial pressure, probably triggering a vigorous sympathetic outflow hyperactivity leading to NSM