RESUMO
Resumen: La programación de un paciente con implante coclear resulta un proceso inicialmente estandarizado, pero que debe individualizarse para cada caso. En la mayoría de las ocasiones, dicho proceso implica el establecimiento de parámetros comunes. Sin embargo, existe un porcentaje de usuarios en los que por alguna circunstancia no es posible establecer niveles adecuados de estimulación, por lo que el médico audiólogo tiene que realizar ajustes en parámetros especiales, como cambio de estrategia, tasa de estimulación, ancho de pulso, entre otros, con el fin de obtener un desempeño auditivo de acuerdo a lo esperado.
Abstract: Programming a patient with cochlear impant follows a standardized and individualized protocol, although there is a percentage of users in which, for some reason, it is not possible to establish appropriate levels of stimulation. In these patients, the audiologist has to make adjustments in some special parameters such as a change in the strategy, stimulation rate, pulse width, among others, in order to obtain an auditory performance as expected.
Assuntos
Humanos , Implantes Cocleares , Implante Coclear/métodos , Audiologistas/organização & administração , Perda Auditiva/cirurgiaRESUMO
There has been controversy between compression of the facial nerve in the cerebellopontine recess and focal demyelination of the facial nerve on the pathogenesis of hemifacial spasm(HFS). To know the pathophysiology of HFS, we performed the facial nerve stimulation test and blink reflex test in 15 patients with HFS. And then we analyzed change of electrophysiologic findings after botulinum toxin in jection. We could not find any differences of latency between affected and unaffected side in facial nerve stimulation test(p>0.05). In the blink reflex test, there were prolonged R1 latency(p<0.01) and occurance of late response on the affected side. There has no change after botulinum toxin injection. These results suggest that there is no facial neuropathy and no electrophysiological evidence of dysfunction in the blink reflex system. Also, there may be two pathophysiologic lesions in HFS. compression and focal demyelination of the facial nerve. And we think that the botulinum toxin is no effect on facial nerve conduction itself.