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Artigo em Inglês | WPRIM | ID: wpr-67983

RESUMO

Stent thromboses due to multifactorial causes including hypercoagulable conditions and high on treatment platelet reactivity (HTPR), which means a low response to anti-platelet therapy, especially clopidogrel. Prasugrel is a third generation thienopyridine and inactive pro-drug requiring metabolic activation in vivo, which improves the rate of HTPR with clopidogrel. This drug is mostly effective, with a potent, fast, and consistent anti-platelet action, but rare cases of inadequate platelet inhibition with prasugrel have been reported. Here we describe the case of a 47-year-old man who presented with a recurrent acute myocardial infarction and ST during an intravascular ultrasound pullback and was resistant to prasugrel, was successfully treated with ticagrelor.


Assuntos
Humanos , Pessoa de Meia-Idade , Ativação Metabólica , Plaquetas , Infarto do Miocárdio , Cloridrato de Prasugrel , Stents , Trombose , Ultrassonografia
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