Memoria e historia desafíos a las prácticas políticas de olvido en américa latina / Memory and history: challenges to the forgotten political practices in latin america

Memoria y olvido han sido, desde la emergencia de nuestra especie en este mundo, significantes que nombran el gran desafío de la supervivencia simbólica en el mundo de la vida. Es la constante lucha por registrar no sólo nuestra propia existencia sino un modo de transitarla, de construirla y vivirla, exorcizando así el fantasma del olvido; porque el olvido es el enemigo de la vida misma, es el nombre de las experiencias perdidas ¿Qué sería de nosotros si olvidáramos las experiencias – más allá de su belleza o dolor - y los aprendizajesque, muchas veces con gran esfuerzo, hemos caminado en este entrelazamiento entre la especie humana y el transcurrir del tiempo?.

Memory and oblivion have been, since the emergence of our species in this world, signifiers that named the great challenge of symbolic survival in the world of life. It is the constantstruggle to record not only our own existence, but a way to move it, build it and live it, thus exorcising the ghost of oblivion; because oblivion is the enemy of life itself, it is the name of the lost experience. What would become of us if we forget experiences - beyond theirbeauty or pain—and the learning that, many times with great effort, we have stepped into this entanglement between the human species and the passing of time?.

Amifostine ameliorates recognition memory defect in acute radiation syndrome caused by relatively low-dose of gamma radiation

This study examined whether amifostine (WR-2721) could attenuate memory impairment and suppress hippocampal neurogenesis in adult mice with the relatively low-dose exposure of acute radiation syndrome (ARS). These were assessed using object recognition memory test, the terminal deoxynucleotidyl transferase-mediated dUTP nick end-labeling assay, and immunohistochemical markers of neurogenesis [Ki-67 and doublecortin (DCX)]. Amifostine treatment (214 mg/kg, i.p.) prior to irradiation significantly attenuated the recognition memory defect in ARS, and markedly blocked the apoptotic death and decrease of Ki-67- and DCX-positive cells in ARS. Therefore, amifostine may attenuate recognition memory defect in a relatively low-dose exposure of ARS in adult mice, possibly by inhibiting a detrimental effect of irradiation on hippocampal neurogenesis.

Spatial memory perfomance of wistar rats exposed to mobile phone

INTRODUCTION: With the tremendous increase in number of mobile phone users world wide, the possible risks of this technology have become a serious concern. OBJECTIVE: We tested the effects of mobile phone exposure on spatial memory performance. MATERIALS AND METHODS: Male Wistar rats (10-12 weeks old) were exposed to 50 missed calls/day for 4 weeks from a GSM (900/1800MHz) mobile phone in vibratory mode (no ring tone). After the experimental period, the animals were tested for spatial memory performance using the Morris water maze test. RESULTS: Both phone exposed and control animals showed a significant decrease in escape time with training. Phone exposed animals had significantly (~3 times) higher mean latency to reach the target quadrant and spent significantly (~2 times) less time in the target quadrant than age- and sex-matched controls. CONCLUSION: Mobile phone exposure affected the acquisition of learned responses in Wistar rats. This in turn points to the poor spatial navigation and the object place configurations of the phone-exposed animals.

[Effect of cell phone radiation [940 MHz] on the learning and memory of Balb/c mice]

The possible risks of radio-frequency electromagnetic fields for human body are a growing concern for our society. In the modern world, the increase of using cell phones is remarkable in all generations especially young people. In addition many users hold phone close to their head. Therefore, we have studied the effect of cell phone radiation [940 MHz] on memory and learning of Balb/C mice which were exposed after delivery. A purpose-designed exposure at a specific absorption for 3h/day from day 1 to 20 after delivery was used. Any stress response in the brain was detected in hippocampus. Behavioral performance began 2.5 month after delivery and testing was conducted in the water maze and the radial maze distinguished working and reference memory performance. After behavioral studies, fetal heads were collected, fixed in 10% paraformaldehyde and paraffine embedded. Microwave-exposed mice were slower than sham-exposed, and cage control in swim speed [WM]. Error Analyses rates reveal significant exposure effect in RAM and MWM. However, in this study the exposed group had not significantly lost their hippocampal CA3 neurons comparing to controls or sham exposed group. Increased time to locate a submerged in water maze, reference memory [entries into unbaited arms], working memory [repeated entries into baited arms], show that acute exposure to pulsed microwaves caused a deficit in spatial reference memory in the mouse