Tumor necrosis factor-alpha [TNF - alpha] gene expression in chronic hepatitis B virus infection
Egyptian Journal of Hospital Medicine [The]. 2015; 60 (July): 324-335
in English
| IMEMR
| ID: emr-173933
ABSTRACT
Background:
Tumor necrosis-alpha [TNF-alpha] is produced by macrophages, neutrophils, T-cells and NK-cells after stimulation. In turn, TNF-alpha can stimulate secretion, increase the expression of adhesion molecules as well as active neutrophils. Hence, it fulfills the role as a principal mediator of cellular immune response and inflammation, and may play an important role in non-cytopathic and cytolytic clearance of hepatitis B virus [HBV]. The clearance of HBV is a complex process which may be influenced by many factors including polymorphisms in the tumor necrosis
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Index:
IMEMR (Eastern Mediterranean)
Main subject:
Polymorphism, Genetic
/
Polymorphism, Restriction Fragment Length
/
Gene Expression
/
Hepatitis B virus
/
Polymerase Chain Reaction
/
Hepatitis B, Chronic
Limits:
Adult
/
Humans
Language:
English
Journal:
Egypt. J. Hosp. Med.
Year:
2015
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