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role of M2000 as an anti-inflammatory agent in toll-like receptor 2/microRNA-155 pathway
Fatemeh, Pourgholi; Mahsa, Hajivalili; Rasoul, Razavi; Shadi, Esmaeili; Behzad, Baradaran; Ali, Movasaghpour; Sanam, Sadreddini; Hamidreza, Goodarzynejad; Abbas, Mirshafiey; Mehdi, Yousefi.
AJMB-Avicenna Journal of Medical Biotechnology. 2017; 9 (1): 8-12
in English | IMEMR | ID: emr-185806
ABSTRACT

Background:

M2000 is a newly designed and safe Non-Steroidal Anti-Inflammatory Drug [NSAID]. The aim of this study was to assess the effects of M2000 on expression levels of Suppressor of Cytokine Signaling-1 [SOCS-1] and Src Homology-2 domain containing inositol-5'-phosphatase 1 [SHIP1] proteins via Toll-Like Receptor [TLR] 2/microRNA-155 pathway
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Index: IMEMR (Eastern Mediterranean) Main subject: Src Homology Domains / MicroRNAs / Toll-Like Receptor 2 / Suppressor of Cytokine Signaling 1 Protein / Iran Limits: Humans Country/Region as subject: Asia Language: English Journal: Avicenna J. Med. Biotechnol. Year: 2017

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Index: IMEMR (Eastern Mediterranean) Main subject: Src Homology Domains / MicroRNAs / Toll-Like Receptor 2 / Suppressor of Cytokine Signaling 1 Protein / Iran Limits: Humans Country/Region as subject: Asia Language: English Journal: Avicenna J. Med. Biotechnol. Year: 2017