De la hipótesis de la trombina a la inflamación. Es una realidad? / From thrombin hypothesis to inflammation. Is it reality?
Arch. cardiol. Méx
; Arch. cardiol. Méx;76(supl.2): S233-S238, abr.-jun. 2006.
Article
in Es
| LILACS
| ID: lil-568813
Responsible library:
BR1.1
ABSTRACT
The classical pathophysiologic concept of the acute coronary syndromes is the coronary artery thrombosis as a consequence of rupture or vulnerable atherosclerotic plaques. Actually, it is also been considered that systemic inflammatory phenomenon play a central role in the plaque instability associated to the atherothrombotic activity of the tissue factor (TF). The thrombotic phenomenon is controlled by tissue factor, stimulating the way of the protease's active receptors (PAR) and cause a negative cycle between inflammation and coagulation.
Full text:
1
Index:
LILACS
Main subject:
Thrombin
/
Inflammation
/
Angina, Unstable
/
Myocardial Infarction
Limits:
Humans
Language:
Es
Journal:
Arch Cardiol Mex
/
Arch. cardiol. Méx
/
Archivos de cardiologia de México
Journal subject:
CARDIOLOGIA
Year:
2006
Type:
Article