Two Cases of Miller Fisher Syndrome without Ataxia
Journal of the Korean Child Neurology Society
;
: 205-210, 2007.
Article
in Korean
| WPRIM
| ID: wpr-127705
ABSTRACT
A classic triad of acute external ophthalmoplegia, areflexia and ataxia characterizes Miller Fisher syndrome(MFS). Diagnosis is based on clinical findings and supported by two laboratory findings; CSF albuminocytological dissociation and serum anti-GQ1b IgG antibody testing. Anti-GQ1b antibody is a key factor in the pathogenesis of Miller Fisher syndrome and a useful marker in laboratory diagnosis. Here we report 2 cases with Miller Fisher syndrome without ataxia, whose major symptom was acute external ophthalmoplegia. Case 1 was associated with preceding Epstein-Barr virus infection and negative anti- GQ1b antibody. Case 2 was positive for the anti-GQ1b antibody. Both received intravenous immunoglobulin and fully recovered within 2 months after the onset of disease.
Full text:
Available
Index:
WPRIM (Western Pacific)
Main subject:
Ataxia
/
Immunoglobulin G
/
Immunoglobulins
/
Ophthalmoplegia
/
Herpesvirus 4, Human
/
Clinical Laboratory Techniques
/
Miller Fisher Syndrome
/
Diagnosis
Type of study:
Diagnostic study
Language:
Korean
Journal:
Journal of the Korean Child Neurology Society
Year:
2007
Type:
Article
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