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Apraxia of Lid Opening after Traumatic Brain Injury: A Delayed Complication
Journal of the Korean Neurological Association ; : 682-685, 2005.
Article in Korean | WPRIM | ID: wpr-199757
ABSTRACT
Apraxia of lid opening (ALO) has been suggested to be a dysfunction of the supranuclear control of the levator palpebrae superioris caused mainly by basal ganglial lesion. The hypometabolism of the medial frontal lobe may be a pathophysiologic mechanism in ALO. We report two ALO patients who developed these symptoms as a delayed complication after traumatic brain injury (TBI). Their MRI showed encephalomalacia in the Rt. medial frontal cortex, which was not shown in initial brain CT scans. Delayed pathologic changes after TBI may contribute to the development of ALO in these cases.
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Full text: Available Index: WPRIM (Western Pacific) Main subject: Apraxias / Brain / Brain Injuries / Magnetic Resonance Imaging / Tomography, X-Ray Computed / Encephalomalacia / Frontal Lobe Limits: Humans Language: Korean Journal: Journal of the Korean Neurological Association Year: 2005 Type: Article

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Full text: Available Index: WPRIM (Western Pacific) Main subject: Apraxias / Brain / Brain Injuries / Magnetic Resonance Imaging / Tomography, X-Ray Computed / Encephalomalacia / Frontal Lobe Limits: Humans Language: Korean Journal: Journal of the Korean Neurological Association Year: 2005 Type: Article