A Case of Licorice Induced Hypokalemic Paralysis with Rhabdomyolysis / 대한내분비학회지
Journal of Korean Society of Endocrinology
;
: 179-182, 2005.
Article
in Korean
| WPRIM
| ID: wpr-87240
ABSTRACT
Prolonged ingestion of licorice can cause hypermineralocorticoidism, with sodium retention, potassium loss and hypertension. Nevertheless, its initial presentation with a very severe degree of hypokalemic paralysis and rhabdomyolysis are exceedingly rare. We describe a patient who experienced hypokalemic paralysis and rhabdomyolysis after licorice ingestion. The patient's initial blood pressure was 160/80mmHg. The major biochemical abnormalities included; hypokalemia(K+ 1.3mEq/L), metabolic alkalosis, with a pH of 7.64, and urine myoglobin > 3000ng/mL. The plasma rennin activity and aldosterone level were suppressed. The 24 hour urine cortisol concentration was normal. The patients, over a 1 month period, had ingested 500g of licorice boiled in water. After quitting the licorice, the hypokalemia and muscle paralysis gradually improved and blood pressure returned to normal
Full text:
Available
Index:
WPRIM (Western Pacific)
Main subject:
Paralysis
/
Plasma
/
Potassium
/
Rhabdomyolysis
/
Sodium
/
Blood Pressure
/
Hydrocortisone
/
Water
/
Chymosin
/
Glycyrrhizic Acid
Limits:
Humans
Language:
Korean
Journal:
Journal of Korean Society of Endocrinology
Year:
2005
Type:
Article
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