Myasthenia in Acquired Neuromyotonia
Journal of Clinical Neurology
;
: 69-71, 2014.
Artículo
en Inglés
| WPRIM
| ID: wpr-113288
ABSTRACT
BACKGROUND:
Acquired neuromyotonia (NMT) forms part of the spectrum of acquired peripheral nerve hyperexcitability syndrome, and is thought to be caused by antibodies to voltage-gated potassium channels (VGKC). Exertional weakness is unusual unless autoimmune myasthenia gravis (MG) is superimposed. CASE REPORT A case of acquired NMT accompanied by exertional weakness without coexistence of seropositive MG is reported herein.CONCLUSIONS:
Clinical and electrophysiological observations suggest that the cholinergic overactivity in NMT can compromise the safety factor sufficiently to cause a defect in neuromuscular junction transmission.
Texto completo:
Disponible
Índice:
WPRIM (Pacífico Occidental)
Asunto principal:
Nervios Periféricos
/
Síndrome de Isaacs
/
Canales de Potasio con Entrada de Voltaje
/
Anticuerpos
/
Miastenia Gravis
/
Unión Neuromuscular
Idioma:
Inglés
Revista:
Journal of Clinical Neurology
Año:
2014
Tipo del documento:
Artículo
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