De la hipótesis de la trombina a la inflamación. Es una realidad? / From thrombin hypothesis to inflammation. Is it reality?
Arch. cardiol. Méx
;
76(supl.2): S233-S238, abr.-jun. 2006.
Article
Dans Espagnol
| LILACS
| ID: lil-568813
ABSTRACT
The classical pathophysiologic concept of the acute coronary syndromes is the coronary artery thrombosis as a consequence of rupture or vulnerable atherosclerotic plaques. Actually, it is also been considered that systemic inflammatory phenomenon play a central role in the plaque instability associated to the atherothrombotic activity of the tissue factor (TF). The thrombotic phenomenon is controlled by tissue factor, stimulating the way of the protease's active receptors (PAR) and cause a negative cycle between inflammation and coagulation.
Texte intégral:
Disponible
Indice:
LILAS (Amériques)
Sujet Principal:
Thrombine
/
Inflammation
/
Angor instable
/
Infarctus du myocarde
Limites du sujet:
Humains
langue:
Espagnol
Texte intégral:
Arch. cardiol. Méx
Thème du journal:
Cardiologie
Année:
2006
Type:
Article
Pays d'affiliation:
Mexique
Institution/Pays d'affiliation:
Instituto Nacional de Cardiología Ignacio Chávez/MX
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