Calcium cycling proteins in heartfailure, cardiomyopathy and arrhythmias
Experimental & Molecular Medicine
;
: 193-203, 2004.
Article
Dans Anglais
| WPRIM
| ID: wpr-217502
ABSTRACT
A growing body of evidence, including studies using genetically engineered mouse models, has shown that Ca2+ cycling and Ca2+ -dependent signaling pathways play a pivotal role in cardiac hypertrophy and heart failure. In addition, recent studies identified that mutations of the genes encoding sarcoplasmic reticulum (SR) proteins cause human cardiomyopathies and lethal ventricular arrhythmias. The regulation of Ca2+ homeostasis via the SR proteins may have potential therapeutic value for heart diseases such as cardiomyopathy, heart failure and arrhythmias.
Texte intégral:
Disponible
Indice:
WPRIM (Pacifique occidental)
Sujet Principal:
Troubles du rythme cardiaque
/
Réticulum sarcoplasmique
/
Protéines de liaison au calcium
/
Animal génétiquement modifié
/
Bas débit cardiaque
/
Canaux calciques
/
Calcium
/
Cardiopathies
/
Mutation
/
Cardiomyopathies
Type d'étude:
Étude pronostique
Limites du sujet:
Animaux
/
Humains
langue:
Anglais
Texte intégral:
Experimental & Molecular Medicine
Année:
2004
Type:
Article
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