Progress of molecular genetics research on rheumatoid arthritis / 中华医学遗传学杂志
Chinese Journal of Medical Genetics
;
(6): 728-733, 2015.
Article
Dans Chinois
| WPRIM
| ID: wpr-287999
ABSTRACT
Rheumatoid arthritis (RA) is a chronic, inflammatory, autoimmune disorder that principally attacks flexible joints and synovia. The precise pathogenesis of RA remains unclear, and genetic factors probably play an important role in its etiology. In addition to genes from human leukocyte antigen (HLA) region, such as HLA-DRB, genes from non-HLA region, such as TIM-3, PTPN22, TRAF1/C5, STAT4, CCR5, PADI4 and FCGR2A may also contribute to its susceptibility. The advance in molecular genetics research on RA is reviewed here.
Texte intégral:
Disponible
Indice:
WPRIM (Pacifique occidental)
Sujet Principal:
Polyarthrite rhumatoïde
/
Récepteurs du fragment Fc des IgG
/
Prédisposition génétique à une maladie
/
Protein Tyrosine Phosphatase, Non-Receptor Type 22
/
Chaines HLA-DRB1
/
Exome
/
Récepteur cellulaire-2 du virus de l'hépatite A
/
Génétique
/
Protéines membranaires
Limites du sujet:
Humains
langue:
Chinois
Texte intégral:
Chinese Journal of Medical Genetics
Année:
2015
Type:
Article
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