Impaired memory and evidence of histopathology in CA1 pyramidal neurons through Injection of Abeta1-42 peptides into the frontal cortices of rat
Basic and Clinical Neuroscience. 2016; 7 (1): 31-42
em Inglês
| IMEMR
| ID: emr-178781
ABSTRACT
Introduction:
Alzheimer's disease [AD] is one of the most common neurodegenerative disorders, which has much benefited from animal models to find the basics of its pathophysiology. In our previous work [Haghani, Shabani, Javan, Motamedi, and Janahmadi, 2012], a non-transgenic rat model of AD was used in electrophysiological studies. However, we did not investigate the histological aspects in the mentioned study
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Índice:
IMEMR (Mediterrâneo Oriental)
Assunto principal:
Fragmentos de Peptídeos
/
Encefalopatias
/
Peptídeos beta-Amiloides
/
Ratos Wistar
/
Doença de Alzheimer
/
Lobo Frontal
/
Melatonina
/
Memória
Tipo de estudo:
Síntese de evidências
Limite:
Animais
Idioma:
Inglês
Revista:
Basic Clin. Neurosci.
Ano de publicação:
2016
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