A Case of Cerebellar Dysfunction After Acute Organotin Poisoning / 대한산업의학회지
Korean Journal of Occupational and Environmental Medicine
;
: 289-292, 2009.
Artigo
em Coreano
| WPRIM
| ID: wpr-177606
ABSTRACT
BACKGROUND:
We performed a follow-up study of an acute organotin poisoning case on the neurological sequelae that remained after 3 years since diagnosis. CASE In the previous study, a 43-year-old male was reported for acute organotin poisoning. After 3 years, the neurological sequelae were studied using neurological examination, cognitive tests, neurobehavioral tests (by CAYSYS 2000(TM)), brain magnetic resonance imaging (MRI), and 18F-fluorodeoxyglucose (18F-FDG) PET/CT. Upon neurological examination and other tests, orientation was improved compared to that 3 years before, but cerebellar ataxia, dysmetria and dysdiadochokinesia remained the same, with little change. Brain MRI showed cerebellar atrophy. 18F-FDG PET/CT revealed mildly decreased metabolic activity in the pons and in both cerebellar hemispheres.DISCUSSION:
This is the first brain MRI study on cerebellar atrophy caused by organotin poisoning. Cerebellar ataxia was confirmed by decreased metabolic activity in 18F-FDG PET/CT.
Texto completo:
DisponíveL
Índice:
WPRIM (Pacífico Ocidental)
Assunto principal:
Orientação
/
Atrofia
/
Encéfalo
/
Ponte
/
Imageamento por Ressonância Magnética
/
Ataxia Cerebelar
/
Doenças Cerebelares
/
Seguimentos
/
Fluordesoxiglucose F18
/
Exame Neurológico
Tipo de estudo:
Estudo observacional
/
Estudo prognóstico
Limite:
Adulto
/
Humanos
/
Masculino
Idioma:
Coreano
Revista:
Korean Journal of Occupational and Environmental Medicine
Ano de publicação:
2009
Tipo de documento:
Artigo
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