Depression after Traumatic Brain Injury
Journal of the Korean Society of Biological Psychiatry
;
: 21-29, 1999.
Artigo
em Coreano
| WPRIM
| ID: wpr-724890
ABSTRACT
Traumatic Brain Injury(TBI) of any severity can result in broad and persisting biopsychosocial sequelae. Depression after TBI occur at a greater frequency than in the general population, with estimates approaching 25% to 50% for major depression, and 155 to 30% for dysthmia. Acute onset depressions are related to lesion location and may have their etiology in biological response of the injured brain, whereas delayed onset depressions may be mediated by psychosocial factors, suggesting psychological reactions as a possible mechanism. Anxious depressions are associated with right hemisphere lesions, whereas major depressions alone are associated with left dorsolateral frontal and left basal ganglia lesions. However, there is insufficient information to postulate a specific neuroanatomic model for TBI-related depression.
Texto completo:
DisponíveL
Índice:
WPRIM (Pacífico Ocidental)
Assunto principal:
Psicologia
/
Gânglios da Base
/
Encéfalo
/
Lesões Encefálicas
/
Depressão
/
Traumatismos Craniocerebrais
Tipo de estudo:
Estudo prognóstico
Idioma:
Coreano
Revista:
Journal of the Korean Society of Biological Psychiatry
Ano de publicação:
1999
Tipo de documento:
Artigo
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