Likelihood of amyloid formation in COVID-19-induced ARDS.

Sinha, Nabodita; Thakur, Ashwani K

Sinha, Nabodita; Thakur, Ashwani K.

Sinha N; Department of Biological Sciences and Bioengineering, Indian Institute of Technology Kanpur, UP 208016, India.
Thakur AK; Department of Biological Sciences and Bioengineering, Indian Institute of Technology Kanpur, UP 208016, India. Electronic address: akthakur@iitk.ac.in.

Trends Microbiol ; 29(11): 967-969, 2021 11.

Article in English | MEDLINE | ID: covidwho-1157751

ABSTRACT

ABSTRACT

Severe coronavirus disease 2019 (COVID-19) infection leads to multifactorial acute respiratory distress syndrome (ARDS), with little therapeutic success. The pathophysiology associated with ARDS or post-ARDS is not yet well understood. We hypothesize that amyloid formation occurring due to protein homeostasis disruption can be one of the complications associated with COVID-19-induced-ARDS.

Subject(s)

Amyloid/metabolism; COVID-19/complications; COVID-19/virology; Respiratory Distress Syndrome/etiology; Respiratory Distress Syndrome/metabolism; SARS-CoV-2; Amyloidosis/etiology; Amyloidosis/metabolism; Amyloidosis/pathology; Animals; Disease Management; Disease Susceptibility; Humans; Respiratory Distress Syndrome/diagnosis

Keywords

COVID-19; SARS-CoV-2; acute respiratory distress syndrome; amyloidosis; amyloids

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Full text: Available Collection: International databases Database: MEDLINE Main subject: Respiratory Distress Syndrome / SARS-CoV-2 / COVID-19 / Amyloid Type of study: Diagnostic study Topics: Long Covid Limits: Animals / Humans Language: English Journal: Trends Microbiol Journal subject: Microbiology Year: 2021 Document Type: Article Affiliation country: J.tim.2021.03.008

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