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COVID-19, Ebola virus disease, and Nipah virus infection reclassification as novel acute immune dysrhythmia syndrome (n-AIDS): potential crucial role for immunomodulators.
Kelleni, Mina T.
  • Kelleni MT; Pharmacology Department, College of Medicine, Minia University, Minya, Egypt. drthabetpharm@yahoo.com.
Immunol Res ; 69(5): 457-460, 2021 10.
Article in English | MEDLINE | ID: covidwho-1345195
ABSTRACT
In this manuscript, COVID-19, Ebola virus disease, Nipah virus infection, SARS, and MERS are suggested to be considered for a novel immunological reclassification as acute onset immune dysrhythmia syndrome (n-AIDS) due to altered monocytic, Th1/Th2, as well as cytokines and chemokines balances. n-AIDs is postulated to be the cause of the acute respiratory distress and multi-inflammatory syndromes which are described with fatal COVID-19, and immunomodulators are suggested to effectively manage the mentioned diseases as well as for other disorders caused by Th1/Th2 imbalance. Meanwhile, para COVID syndrome is suggested to describe various immune-related complications, whether before or after recovery, and to embrace a potential of a latent infection, that might be discovered later, as occurred with Ebola virus disease. Finally, our hypothesis has evolved out of our real-life practice that uses immunomodulatory drugs to manage COVID-19 safely and effectively.
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Full text: Available Collection: International databases Database: MEDLINE Main subject: Cytokines / Hemorrhagic Fever, Ebola / Henipavirus Infections / COVID-19 Type of study: Experimental Studies Limits: Humans Language: English Journal: Immunol Res Journal subject: Allergy and Immunology Year: 2021 Document Type: Article Affiliation country: S12026-021-09219-y

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Full text: Available Collection: International databases Database: MEDLINE Main subject: Cytokines / Hemorrhagic Fever, Ebola / Henipavirus Infections / COVID-19 Type of study: Experimental Studies Limits: Humans Language: English Journal: Immunol Res Journal subject: Allergy and Immunology Year: 2021 Document Type: Article Affiliation country: S12026-021-09219-y