Mucins produced by type II pneumocyte: culprits in SARS-CoV-2 pathogenesis.

Huang, Bo

Huang, Bo.

Huang B; Department of Immunology & National Key Laboratory of Medical Molecular Biology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences (CAMS) & Peking Union Medical College, Beijing, China. tjhuangbo@hotmail.com.

Cell Mol Immunol ; 18(7): 1823-1825, 2021 07.

Article in English | MEDLINE | ID: covidwho-1387307

Subject(s)

Alveolar Epithelial Cells/virology; COVID-19/virology; Cytokines/metabolism; Macrophage Activation; Macrophages, Alveolar/virology; Mucins/metabolism; Paracrine Communication; SARS-CoV-2/pathogenicity; Alveolar Epithelial Cells/metabolism; COVID-19/metabolism; COVID-19/physiopathology; Disease Progression; Host-Pathogen Interactions; Humans; Macrophages, Alveolar/metabolism; Signal Transduction

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Full text: Available Collection: International databases Database: MEDLINE Main subject: Cytokines / Macrophages, Alveolar / Paracrine Communication / Alveolar Epithelial Cells / SARS-CoV-2 / COVID-19 / Macrophage Activation / Mucins Limits: Humans Language: English Journal: Cell Mol Immunol Journal subject: Allergy and Immunology Year: 2021 Document Type: Article Affiliation country: S41423-021-00714-8

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