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P53 in endothelial function and unfolded protein response regulation.
Kubra, Khadeja-Tul; Barabutis, Nektarios.
  • Kubra KT; School of Basic Pharmaceutical and Toxicological Sciences, College of Pharmacy, University of Louisiana Monroe, Monroe, Louisiana, USA.
  • Barabutis N; School of Basic Pharmaceutical and Toxicological Sciences, College of Pharmacy, University of Louisiana Monroe, Monroe, Louisiana, USA.
Cell Biol Int ; 46(12): 2257-2261, 2022 Dec.
Article in English | MEDLINE | ID: covidwho-1999839
ABSTRACT
Vascular barrier dysfunction due to endothelial hyperpermeability has been associated with the pathophysiology of sepsis and severe lung injury, which may inflict acute respiratory distress syndrome (ARDS). Our group is focused on the mechanisms operating towards the regulation of endothelial permeability, to contribute in the development of efficient and targeted countermeasures against ARDS. Unfortunately, the number of ARDS-related deaths in the intensive care units has dramatically increased during the COVID-19 era. The findings described herein inform the corresponding scientific and medical community on the relation of P53 and stress responses in barrier function.
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Full text: Available Collection: International databases Database: MEDLINE Main subject: Respiratory Distress Syndrome / Sepsis / COVID-19 Limits: Humans Language: English Journal: Cell Biol Int Year: 2022 Document Type: Article Affiliation country: Cbin.11891

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Full text: Available Collection: International databases Database: MEDLINE Main subject: Respiratory Distress Syndrome / Sepsis / COVID-19 Limits: Humans Language: English Journal: Cell Biol Int Year: 2022 Document Type: Article Affiliation country: Cbin.11891