Is susceptibility to severe COVID-19 disease an inborn error of metabolism?

Clayton, Peter T

Clayton, Peter T.

Clayton PT; Genetics and Genomic Medicine, UCL Great Ormond Street Institute of Child Health, London, UK.

J Inherit Metab Dis ; 43(5): 906-907, 2020 09.

Article in English | MEDLINE | ID: covidwho-625617

Subject(s)

Adaptor Proteins, Signal Transducing/metabolism; Betacoronavirus/physiology; Coronavirus Infections; Membrane Glycoproteins/metabolism; Pandemics; Peptidyl-Dipeptidase A/metabolism; Pneumonia, Viral; Proline/metabolism; Angiotensin-Converting Enzyme 2; COVID-19; Coronavirus Infections/immunology; Coronavirus Infections/metabolism; Coronavirus Infections/virology; Genome-Wide Association Study; Humans; Membrane Transport Proteins/metabolism; Mutation; Pneumonia, Viral/etiology; Pneumonia, Viral/immunology; Pneumonia, Viral/metabolism; Pneumonia, Viral/virology; SARS-CoV-2

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Full text: Available Collection: International databases Database: MEDLINE Main subject: Pneumonia, Viral / Proline / Membrane Glycoproteins / Coronavirus Infections / Peptidyl-Dipeptidase A / Adaptor Proteins, Signal Transducing / Pandemics / Betacoronavirus Limits: Humans Language: English Journal: J Inherit Metab Dis Year: 2020 Document Type: Article Affiliation country: Jimd.12280

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