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Is the Reason of Increased D-Dimer Levels in COVID-19 Because of ACE-2-Induced Apoptosis in Endothelium?
Guler, Nil; Siddiqui, Fakiha; Fareed, Jawed.
  • Guler N; Department of Hematology, Medical School, Pamukkale University, Denizli, Turkey.
  • Siddiqui F; Department of Pathology, Cardiovascular Research Institute, Loyola University Chicago, Health Sciences Division, Maywood, IL, USA.
  • Fareed J; Department of Pathology and Laboratory Medicine, Department of Pharmacology and Neuroscience, Cardiovascular Research Institute, Loyola University Chicago, Health Sciences Division, Maywood, IL, USA.
Clin Appl Thromb Hemost ; 26: 1076029620935526, 2020.
Article in English | MEDLINE | ID: covidwho-643163
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Full text: Available Collection: International databases Database: MEDLINE Main subject: Pneumonia, Viral / Receptors, Virus / Fibrin Fibrinogen Degradation Products / Endothelium, Vascular / Apoptosis / Coronavirus Infections / Peptidyl-Dipeptidase A / Thrombophilia / Host-Pathogen Interactions / Betacoronavirus Topics: Long Covid Limits: Animals / Humans Language: English Journal: Clin Appl Thromb Hemost Journal subject: Vascular Diseases Year: 2020 Document Type: Article Affiliation country: 1076029620935526

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Full text: Available Collection: International databases Database: MEDLINE Main subject: Pneumonia, Viral / Receptors, Virus / Fibrin Fibrinogen Degradation Products / Endothelium, Vascular / Apoptosis / Coronavirus Infections / Peptidyl-Dipeptidase A / Thrombophilia / Host-Pathogen Interactions / Betacoronavirus Topics: Long Covid Limits: Animals / Humans Language: English Journal: Clin Appl Thromb Hemost Journal subject: Vascular Diseases Year: 2020 Document Type: Article Affiliation country: 1076029620935526