Hypothetical dysfunction of the epithelial sodium channel may justify neurohumoral blockade in coronavirus disease 2019.

Szabó, Gábor Tamás; Kiss, Attila; Csanádi, Zoltán; Czuriga, Dániel

Szabó, Gábor Tamás; Kiss, Attila; Csanádi, Zoltán; Czuriga, Dániel.

Szabó GT; Division of Cardiology, Department of Cardiology, Faculty of Medicine, University of Debrecen, 22 Móricz Zs. krt., Debrecen, H-4032, Hungary.
Kiss A; Center for Biomedical Research, Medical University of Vienna, Vienna, Austria.
Csanádi Z; Division of Cardiology, Department of Cardiology, Faculty of Medicine, University of Debrecen, 22 Móricz Zs. krt., Debrecen, H-4032, Hungary.
Czuriga D; Division of Cardiology, Department of Cardiology, Faculty of Medicine, University of Debrecen, 22 Móricz Zs. krt., Debrecen, H-4032, Hungary.

ESC Heart Fail ; 8(1): 171-174, 2021 02.

Article in English | MEDLINE | ID: covidwho-932427

Subject(s)

COVID-19/metabolism; Epithelial Sodium Channels/metabolism; Neurotransmitter Agents/antagonists & inhibitors; SARS-CoV-2/metabolism; COVID-19/physiopathology; Epithelial Sodium Channels/drug effects; Furin/deficiency; Furin/metabolism; Humans; Renin-Angiotensin System; Spike Glycoprotein, Coronavirus/metabolism

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Full text: Available Collection: International databases Database: MEDLINE Main subject: Neurotransmitter Agents / Epithelial Sodium Channels / SARS-CoV-2 / COVID-19 Limits: Humans Language: English Journal: ESC Heart Fail Year: 2021 Document Type: Article Affiliation country: Ehf2.13078

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