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1.
Differential effects of putative N-glycosylation sites in human Tau on Alzheimer's disease-related neurodegeneration.
Cell Mol Life Sci
; 78(5): 2231-2245, 2021 Mar.
Artículo
en Inglés
| MEDLINE | ID: mdl-32926180
2.
Cl-NQTrp Alleviates Tauopathy Symptoms in a Model Organism through the Inhibition of Tau Aggregation-Engendered Toxicity.
Neurodegener Dis
; 17(2-3): 73-82, 2017.
Artículo
en Inglés
| MEDLINE | ID: mdl-27760426
3.
The contribution of individual residues of an aggregative hexapeptide derived from the human γD-crystallin to its amyloidogenicity.
Int J Biol Macromol
; 201: 182-192, 2022 Mar 15.
Artículo
en Inglés
| MEDLINE | ID: mdl-34998884
4.
Inhibition of amyloid fibrillation of γD-crystallin model peptide by the cochineal Carmine.
Int J Biol Macromol
; 169: 342-351, 2021 Feb 01.
Artículo
en Inglés
| MEDLINE | ID: mdl-33347930
5.
An amyloidogenic hexapeptide from the cataract-associated γD-crystallin is a model for the full-length protein and is inhibited by naphthoquinone-tryptophan hybrids.
Int J Biol Macromol
; 157: 424-433, 2020 Aug 15.
Artículo
en Inglés
| MEDLINE | ID: mdl-32302630
6.
Naphthoquinone-Tryptophan Hybrid Inhibits Aggregation of the Tau-Derived Peptide PHF6 and Reduces Neurotoxicity.
J Alzheimers Dis
; 51(1): 165-78, 2016.
Artículo
en Inglés
| MEDLINE | ID: mdl-26836184
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